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RecruitingNCT07843095ProteinUpdated Sep 25, 2026

Protein Supplementation During Luteal Phase to Stop Protein Degradation

An interventional study of protein supplement and Placebo in Muscle Atrophy, sponsored by Sam Houston State University. Recruiting at 2 sites in United States. Open to female participants aged 18 Years to 35 Years, including healthy volunteers. Per ClinicalTrials.gov, last updated 2026-09-25.

Sponsored by Sam Houston State University · Not applicable, Interventional, and Treatment

Phase
Not applicable
Study type
Interventional
Enrollment
34
Allocation
Randomized
Ages
18 Years to 35 Years
Sex
Female
01

Study summary

The purpose of this research is to determine if increasing 12% of daily protein intake during the luteal phase mitigates the potential catabolic effects of progesterone on skeletal muscle. We anticipate there will be an advantageous, decreased catabolic effect with increasing protein intake during the luteal phase.

By doing this study, we hope to learn if millions of women would benefit from additional protein supplementation during the luteal phase, which may lead to decreased skeletal muscle atrophy over time. Your participation in this research will last about 4 months.

Read the detailed description

The physiologic effects of the female menstrual cycle have been shown to cause many changes to metabolic processes throughout the body depending on the phase of the cycle. For example, during the follicular phase, increase in estrogen aids in arterial vasodilation, lipid metabolism, neuroprotection, and maintaining bone density (Gruber et al. 2002). During the luteal phase, the increase in progesterone aids in thermoregulation, increased respiration, and basal insulin levels (Taraborrelli 2015). However, there is conflicting evidence regarding net progesterone effects on skeletal muscle. Some studies state that progesterone has an anabolic effect while others suggest the contrary. Despite the discrepancies with current literature, Sims et al. recommends female athletes increase their protein intake by 12% during the luteal phase of their menstrual cycle to potentially counteract the protein catabolic effects of progesterone on skeletal muscle (2023). The basis of this conclusion was based on invitro and in vivo studies showing decreases amino acids during the luteal phase (Faustmann et al. 2018 \& Draper et al. 2018)

To our knowledge, no study exists that analyzes protein degradation with protein supplementation increase during the luteal phase. The purpose of this research is to determine if increasing 12% of daily protein intake during the luteal phase mitigates the potential catabolic effects of progesterone on skeletal muscle. This research could help determine if millions of women would benefit from additional protein supplementation during the luteal phase, which may lead to decreased skeletal muscle atrophy over time. We anticipate there will be an advantageous, decreased catabolic effect with increasing protein intake during the luteal phase.

02

Conditions studied

  • Muscle Atrophy

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03

Who can participate

Ages eligible
18 Years to 35 Years
Sexes eligible
Female
Accepts healthy volunteers
Yes

Inclusion criteria

  • Eumenorrheic
  • 18 to 35 years old

Exclusion criteria

Exclusion Criteria:

  • Pregnant or lactating (These individuals will not have a regular menstrual cycle) in the previous six months
  • Vegetarian (protein supplementation is from animal origin)
  • Contraceptive use, any hormone based contraceptive use for 6 months prior to enrollment
  • Use of illicit drugs in previous 6 months.
  • Non-tobacco user
  • Gasterointestinal disorders, liver disease, infectious disease, cardiovascular, pulmonary, renal, immune, metabolic, cancer
  • Medications: corticosteroids, chronic NSAIDS, high strength acne, testosterone
  • Diagonsed PCOS, endometrosis, ovarian insufficency, or other menstrual cycle disorder.
04

Study design

Phase
Not applicable
Primary purpose
Treatment
Allocation
Randomized
Intervention model
Crossover assignment
Masking
Quadruple (Participant, Care provider, Investigator, Outcomes assessor)
Enrollment
34 participants (estimated)

Study arms

  • Active comparator
    A

    Will receive protein supplement

    Dietary Supplement: protein supplement

  • Placebo comparator
    B

    Will receive placebo supplement

    Dietary Supplement: Placebo

Interventions

  • Dietary supplementprotein supplement

    Protein supplementation during progesterone peak to stop protein degradation effect.

    Also known as: A

  • Dietary supplementPlacebo

    Will receive placebo supplementation

    Also known as: B

05

What researchers measure

Primary outcomes

  1. Skeletal Muscle Protein Degradation

    Primary outcomes will be changes in urinary N-fragment of titin and serum 3-methylhistidine between supplementation and placebo conditions. Paired t-tests or linear mixed-effects models will be used to account for within-subject crossover effects. Hormone levels will be included as covariates. Statistical significance will be set at p \< 0.05.

    Time frame: 48 hours

Secondary outcomes

  1. Decrease protein degradation

    We would like to home in on protein intake and degradation to quantify the amount of protein loss seen during the luteal phase, the point at which protein degradation is deemed to be the highest. Our specific aims for this project will be to 1) quantify protein degradation at the time of the luteal phase and 2) determine whether targeted protein supplementation during the luteal phase will reduce measured protein degradation. We anticipate there will be an advantageous, decreased catabolic effect with increasing protein intake during the luteal phase. To measure this, we will use Blood and Urine samples during the menstrual cycle. Blood Samples: Blood will be collected in serum separator tubes (SST) Serum will be analyzed using enzyme-linked immunosorbent assay (ELISA) methods to measure: 3-Methylhistidine Progesterone Urine Samples: Urine samples will be analyzed using ELISA methods to measure: N-fragment of Titin Creatin

    Time frame: 48 hours

06

Study locations

1 of 2 sites recruiting
  • SHSU College of Osteopathic Medicine
    Conroe, Texas 77304, United States
    • Kyndal Kutac · Contact · kbk016@shsu.edu · 979-743-1135
    • Damian Chiandussi, MD · Contact · dac150@shsu.edu · 936-202-5279
    • Kyndal Kutac · Sub investigator
    • Maniola Olawoye · Sub investigator
    • Maddison Vrazel · Sub investigator
    • Michelle Greene · Sub investigator
    • Damian Chiandussi · Sub investigator
    • Patrick Davis, PhD · Principal investigator
    Recruiting
  • SHSU Physician's Clinic
    Conroe, Texas 77304, United States
    • Kyndal Kutac · Contact · kbk016@shsu.edu · 979-743-1135
    • Damian Chiandussi, MD · Contact · dac150@shsu.edu · 936-202-5279
    • Patrick Davis, PhD · Principal investigator
    Not yet recruiting
07

References and documents

Publications

  • Gruber, C. J., Tschugguel, W., Schneeberger, C., & Huber, J. C. (2002). Production and Actions of Estrogens. New England Journal of Medicine, 346, 340-352. https://doi.org/10.1056/nejmra000471 Taraborrelli, S. (2015). Physiology, production and action of progesterone. In Acta Obstetricia et Gynecologica Scandinavica (Vol. 94, pp. 8-16). Taylor and Francis Ltd. https://doi.org/10.1111/aogs.12771 Sims, S. T., Kerksick, C. M., Smith-Ryan, A. E., Janse de Jonge, X. A. K., Hirsch, K. R., Arent, S. M., Hewlings, S. J., Kleiner, S. M., Bustillo, E., Tartar, J. L., Starratt, V. G., Kreider, R. B., Greenwalt, C., Rentería, L. I., Ormsbee, M. J., VanDusseldorp, T. A., Campbell, B. I., Kalman, D. S., & Antonio, J. (2023). International society of sports nutrition position stand: nutritional concerns of the female athlete. Journal of the International Society of Sports Nutrition, 20(1). https://doi.org/10.1080/15502783.2023.2204066 Faustmann, G., Meinitzer, A., Magnes, C., Tiran, B., Obermayer-Pietsch, B., Gruber, H. J., Ribalta, J., Rock, E., Roob, J. M., & Winklhofer-Roob, B. M. (2018). Progesterone-associated arginine decline at luteal phase of menstrual cycle and associations with related amino acids and nuclear factor kB activation. PLoS ONE, 13(7). https://doi.org/10.1371/journal.pone.0200489 Draper, C. F., Duisters, K., Weger, B., Chakrabarti, A., Harms, A. C., Brennan, L., Hankemeier, T., Goulet, L., Konz, T., Martin, F. P., Moco, S., & van der Greef, J. (2018). Menstrual cycle rhythmicity: metabolic patterns in healthy women. Scientific Reports, 8. https://doi.org/10.1038/s41598-018-32647-0

Individual participant data

Plan to share: Undecided

08

Registry details

Key details

Study ID
NCT07843095
Lead sponsor
Sam Houston State University
Responsible party
Sponsor
First posted
Sep 25, 2026
Start date
Aug 4, 2026
Primary completion
Dec 17, 2026 (estimated)
Completion
Jan 29, 2027 (estimated)
Last update
Sep 25, 2026

Study contacts

Kyndal Kutac
Contact
kbk016@shsu.edu
979-743-1135
Damian Chiandussi, MD
Contact
dac150@shsu.edu
Patrick Davis, PhD
principal investigator · SHSU COM

Oversight

Data monitoring committee
No
FDA-regulated drug
No
FDA-regulated device
No
View the source record on ClinicalTrials.gov ↗

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