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RecruitingNCT07534397Updated Sep 16, 2026

Treatment of Cognitive and Sensorimotor Deficits in Parkinson's Disease With High Definition Transcranial Direct Current Stimulation

An interventional study of transcranial direct current stimulation and Sham transcranial direct current stimulation in PARKINSON DISEASE (Disorder), sponsored by The University of Texas at Dallas. Recruiting at 1 site in United States. Open to participants aged 50 Years to 90 Years. Per ClinicalTrials.gov, last updated 2026-09-16.

Sponsored by The University of Texas at Dallas · Not applicable, Interventional, and Treatment

Phase
Not applicable
Study type
Interventional
Enrollment
20
Allocation
Randomized
Ages
50 Years to 90 Years
Sex
All
01

Study summary

The purpose of this research study is to examine the effects of transcranial Direct Current Stimulation (tDCS) on verbal retrieval and cognition and sensorimotor control and to determine if tDCS can be used as a way to improve retrieval, sensory, and motor abilities in individuals with Parkinson's disease (PD).

Read the detailed description

Objective: Based on models of semantic memory retrieval developed, under which the pre-supplementary motor area (preSMA) of the dorsomedial frontal lobes play an important role, the study will examine the treatment of cognitive deficits in PD with HD tDCS applied to the preSMA to improve function in neural circuits supporting verbal retrieval. In addition, as the preSMA is implicated in motor planning and sequencing, the study also will explore whether HD tDCS applied to the preSMA will have an effect on speech and gait.

Specific Aims:

  1. Examine the therapeutic effects on verbal retrieval function by modulating the preSMA using HD tDCS.
  2. Examine the therapeutic effects on speech production and processing by modulating the preSMA using HD tDCS
  3. Examine the therapeutic effects on gait and balance by modulating the preSMA using HD tDCS

Hypotheses:

  1. : Active HD tDCS (1 ma anodal HD tDCS to the preSMA compared to sham) will improve verbal fluency (i.e, phonemic and category fluency on Controlled Word Association Test) as primary outcome measures.
  2. : Active HD tDCS (1 ma anodal HD tDCS to the preSMA compared to sham) will improve speech sequencing.
  3. : Active HD tDCS (1 ma anodal HD tDCS to the preSMA compared to sham) will improve general motor sequencing
02

Conditions studied

  • PARKINSON DISEASE (Disorder)

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Keywords

  • transcranial direct current stimulation (tDCS)
  • Parkinson's Disease
  • electroencephalography (EEG)
  • presupplementary motor area (preSMA)
  • verbal memory
  • speech sequencing
  • motor sequencing
03

Who can participate

Ages eligible
50 Years to 90 Years
Sexes eligible
All
Accepts healthy volunteers
No

Inclusion criteria

  • Diagnosed with PD having a verbal fluency deficit based on neuropsychological test (T-score \< 40 or a T-score \<-1.0 SD below the average T-score): Semantic Object Retrieval Test (SORT), COWAT (both letter and category fluency), Boston Naming Test (BNT), Rey Auditory Verbal Learning Test (RAVLT)
  • 50 - 90 years old
  • Capable of understanding and signing an informed consent (able to answer consent comprehension questions)
  • Fluent in speaking and reading English

Exclusion criteria

Exclusion Criteria:

A potentially study-confounding, tDCS-contraindicated, or EEG-contraindicated psychological, neuropsychiatric, neurological, or other medical issues:

  • Montreal Cognitve Assessment (MOCA) score \<23, unless an accompanying study partner/caregiver is with them and we can obtain the written consent of both the participant and the participant's accompanying study partner/caregiver (i.e., the participant's spouse, adult child, parent, or adult sibling);
  • history of seizures;
  • unexplained episodes of loss of consciousness (as these may be related to brain alterations or epilepsy);
  • suffering from severe or frequent headaches;
  • unstable or uncontrolled neuropsychiatric illness;
  • severe traumatic brain injury (based on the Ohio State TBI Identification; Method);
  • brain tumor; stroke; present drug abuse/misuse;
  • brain tumor;
  • serious or life-threatening diseases, including congestive heart failure, chronic obstructive pulmonary disease, or active malignancy;
  • Huntington's disease;
  • stroke;
  • cranial implants or skull defects that affect tDCS administration;
  • implanted brain medical devices, including, deep brain stimulators (DBS);
  • implanted pacemakers;
  • any electrically, magnetically, or mechanically activated implants;
  • cardiac, neural, or medication implants;
  • vascular clips or other electrically sensitive support systems in the brain;
  • damaged skin at the sites of stimulation (the device should only be used on healthy, intact skin, as wounds may alter resistance to current);
  • skin conditions such as dermatitis, psoriasis, or eczema;
  • pregnant women;
  • diagnosis of just dysarthria;

Use of medications that interact with or potentially interact with tDCS or EEG effects:

  • anti-convulsants;
  • carbamazepine;
  • sulpiride;
  • pergolide;
  • lorazepam;
  • rivastigmine;
  • dextromethorphan;
  • D-cycloserine;
  • flunarizine;
  • ropinirole;
  • citalopram;
  • stimulants;

Additionally, non-English speakers will be excluded because not all of the screening forms, questionnaires, and tests are available in languages other than English.

04

Study design

Phase
Not applicable
Primary purpose
Treatment
Allocation
Randomized
Intervention model
Parallel assignment
Masking
Quadruple (Participant, Care provider, Investigator, Outcomes assessor)
Enrollment
20 participants (estimated)

Study arms

  • Experimental
    Transcranial direct current stimulation

    Transcranial direct current stimulation will be delivered via a Neuroelectrics Starstim tES. Stimulation will consist of 1 milliamp stimulation, with anodal stimulation delivered at electrode Fz (International 10/10 System for electroencephalography electrode placement) and electrodes F7, FP1, FP2, and F8 as returns. All electrodes are 1 cm diameter Ag/AgCl electrodes and make contact with the scalp via connective gel. Stimulation will linearly ramp up from 0 milliamps to 1 milliamp over 60 seconds, then remain at 1 milliamp of stimulation over 20 minutes, and finally ramping down at to 0 milliamps over 60 seconds. Other Names: tDCS 1 milliamp tDCS High definition tDCS High definition transcranial direct current stimulator, Neuroelectrics Starstim tES, SN E20200930-10

    Device: transcranial direct current stimulation

  • Sham comparator
    Sham transcranial direct current stimulation

    Sham transcranial direct current stimulation will be delivered via a Neuroelectrics Starstim tES. The sham setup will consist of anodal electrode Fz (International 10/10 System for electroencephalography electrode placement) and electrodes F7, FP1, FP2, and F8 as returns. All electrodes are 1 cm diameter Ag/AgCl electrodes and make contact with the scalp via connective gel. Stimulation will linearly ramp up from 0 milliamps to 1 milliamp over 60 seconds, ramp down to 0 milliamps over 60 seconds and then be left off for 20 minutes.

    Device: Sham transcranial direct current stimulation

Interventions

  • Devicetranscranial direct current stimulation

    Transcranial direct current stimulation will be delivered via a Neuroelectrics Starstim tES. Stimulation will consist of 1 milliamp stimulation, with anodal stimulation delivered at electrode Fz (International 10/10 System for electroencephalography electrode placement) and electrodes F7, FP1, FP2, and F8 as returns. All electrodes are 1 cm diameter Ag/AgCl electrodes and make contact with the scalp via connective gel. Stimulation will linearly ramp up from 0 milliamps to 1 milliamp over 60 seconds, then remain at 1 milliamp of stimulation over 20 minutes, and finally ramping down at to 0 milliamps over 60 seconds.

    Also known as: transcranial electric stimulation, tDCS, tES

  • DeviceSham transcranial direct current stimulation

    Sham transcranial direct current stimulation will be delivered via a Neuroelectrics Starstim tES. The sham setup will consist of anodal electrode Fz (International 10/10 System for electroencephalography electrode placement) and electrodes F7, FP1, FP2, and F8 as returns. All electrodes are 1 cm diameter Ag/AgCl electrodes and make contact with the scalp via connective gel. Stimulation will linearly ramp up from 0 milliamps to 1 milliamp over 60 seconds, ramp down to 0 milliamps over 60 seconds and then be left off for 20 minutes.

    Also known as: sham tDCS, Sham trascranial electric stimulation, sham tES

05

What researchers measure

Primary outcomes

  1. Treatment group differences in change from Baseline to 1-week Post-Treatment on Category Fluency

    valuation of treatment group differences in change on Category Fluency from baseline to 1-week post-treatment. Metric: Number of Correct Items Generated (minimum=0 words; no maximum)

    Time frame: Outcome measures will be assessed as change over a period of 6 weeks: Change from baseline to 1-week Post-Treatment

  2. Treatment group differences in change from Baseline to 2-months Post-Treatment on Category Fluency

    Evaluation of treatment group differences in change on Category Fluency from baseline to 2-months post-treatment. Metric: Number of Correct Items Generated (minimum=0 words; no maximum)

    Time frame: Outcome measures will be assessed as change over a period of 13 weeks: Change from baseline to 2-month Post-Treatment

  3. Treatment group differences in change from Baseline to 1-week Post-Treatment on Phonemic Fluency

    Evaluation of treatment group differences in change on Phonemic Fluency from baseline to 1-week post-treatment. Metric: Number of Correct Items Generated (minimum=0 words; no maximum)

    Time frame: Outcome measures will be assessed as change over a period of 6 weeks: Change from baseline to 1-week Post-Treatment

  4. Treatment group differences in change from Baseline to 2-months Post-Treatment on Phonemic Fluency

    Evaluation of treatment group differences in change on Phonemic Fluency from baseline to 2-months post-treatment. Metric: Number of Correct Items Generated (minimum=0 words; no maximum)

    Time frame: Outcome measures will be assessed as change over a period of 13 weeks: Change from baseline to 2-months Post-Treatment

Secondary outcomes

  1. Treatment group differences in change from Baseline to 1-week Post-treatment on Speaking Rate during overt reading

    Evaluation of treatment group differences in change in speaking rate from baseline to 1-week post-treatment. Metric: syllables per second on a passage reading task (minimum=0; no maximum).

    Time frame: Outcome measures will be assessed as change over a period of 6 weeks: Change from baseline to 1-week Post-Treatment

  2. Treatment group differences in change from Baseline to 2-months Post-Treatment on Speaking Rate during overt reading

    Evaluation of treatment group differences in change on speaking rate from baseline to 2-months post-treatment. Metric: Syllables per second during a reading passage (minimum=0; no maximum).

    Time frame: Outcome measures will be assessed as change over a period of 13 weeks: change from baseline to 2-months Post-Treatment

  3. Treatment group differences in change from Baseline to 1-week Post-treatment on Speaking Rate during self-generated speech

    Evaluation of treatment group differences in change in speaking rate from baseline to 1-week post-treatment. Metric: Syllables per second during self-generated speech (minimum=0; no maximum).

    Time frame: Outcome measures will be assessed as change over a period of 6 weeks: Change from baseline to 1-week Post-Treatment

  4. Treatment group differences in change from Baseline to 2-months Post-Treatment on Speaking Rate during self-generated speech

    Evaluation of treatment group differences in change on speaking rate from baseline to 2-months post-treatment. Metric: Syllables per second during self-generated speech (minimum=0; no maximum).

    Time frame: Outcome measures will be assessed as change over a period of 13 weeks: change from baseline to 2-months Post-Treatment

  5. Treatment group differences in change from Baseline to 1-week Post-treatment on Movement Disorder Society-sponsored Unified Parkinson's Disease Rating Scale Part III score.

    Evaluation of treatment group differences in change in Movement Disorder Society-sponsored Unified Parkinson's Disease Rating Scale Part III score from baseline to 1-week post-treatment. Metric: Movement Disorder Society-sponsored Unified Parkinson's Disease Rating Scale Part III score (minimum=0; maximum=132)

    Time frame: Outcome measures will be assessed as change over a period of 6 weeks: Change from baseline to 1-week Post-Treatment

  6. Treatment group differences in change from Baseline to 2-month Post-treatment on Movement Disorder Society-sponsored Unified Parkinson's Disease Rating Scale Part III score.

    Evaluation of treatment group differences in change in Movement Disorder Society-sponsored Unified Parkinson's Disease Rating Scale Part III score from baseline to 2-month post-treatment. Metric: Movement Disorder Society-sponsored Unified Parkinson's Disease Rating Scale Part III score (minimum=0; maximum=132)

    Time frame: Outcome measures will be assessed as change over a period of 13 weeks: Change from baseline to 2-month Post-Treatment

  7. Treatment group differences in change from Baseline to 1-week Post-treatment on Speaking Rhythm during overt reading

    Evaluation of treatment group differences in change in speaking rhythm from baseline to 1-week post-treatment. Metric: coefficient of variation of syllable duration on a passage reading task (minimum=0; no maximum).

    Time frame: Outcome measures will be assessed as change over a period of 6 weeks: Change from baseline to 1-week Post-Treatment

  8. Treatment group differences in change from Baseline to 2-months Post-Treatment on Speaking Rhythm during overt reading

    Evaluation of treatment group differences in change on speaking rhythm from baseline to 2-months post-treatment. Metric: Coefficient of variation of syllable duration during a reading passage (minimum=0; no maximum).

    Time frame: Outcome measures will be assessed as change over a period of 13 weeks: change from baseline to 2-months Post-Treatment

  9. Treatment group differences in change from Baseline to 1-week Post-treatment on Speaking Rhythm during self-generated speech

    Evaluation of treatment group differences in change in speaking rhythm from baseline to 1-week post-treatment. Metric: Coefficient of variation of syllable duration during self-generated speech (minimum=0; no maximum).

    Time frame: Outcome measures will be assessed as change over a period of 6 weeks: Change from baseline to 1-week Post-Treatment

  10. Treatment group differences in change from Baseline to 2-months Post-Treatment on Speaking Rhythm during self-generated speech

    Evaluation of treatment group differences in change on speaking rhythm from baseline to 2-months post-treatment. Metric: Coefficient of variation of syllable duration during self-generated speech (minimum=0; no maximum).

    Time frame: Outcome measures will be assessed as change over a period of 13 weeks: change from baseline to 2-months Post-Treatment

  11. Treatment group differences in change from Baseline to 1-week Post-treatment on Speaking Pause Time during overt reading

    Evaluation of treatment group differences in change in speaking pause time from baseline to 1-week post-treatment. Metric: Pause time as percentage of total speaking time on a passage reading task (minimum=0%; maximum=100%).

    Time frame: Outcome measures will be assessed as change over a period of 6 weeks: Change from baseline to 1-week Post-Treatment

  12. Treatment group differences in change from Baseline to 2-months Post-Treatment on Speaking Pause Time during overt reading

    Evaluation of treatment group differences in change on speaking pause time from baseline to 2-months post-treatment. Metric: Pause time as percentage of total speaking time during a reading passage (minimum=0%; maximum=100%).

    Time frame: Outcome measures will be assessed as change over a period of 13 weeks: change from baseline to 2-months Post-Treatment

  13. Treatment group differences in change from Baseline to 1-week Post-treatment on Speaking Pause Time during self-generated speech

    Evaluation of treatment group differences in change in speaking pause time from baseline to 1-week post-treatment. Metric: Pause time as percentage of total speaking time during self-generated speech (minimum=0%; maximum=100%).

    Time frame: Outcome measures will be assessed as change over a period of 6 weeks: Change from baseline to 1-week Post-Treatment

  14. Treatment group differences in change from Baseline to 2-months Post-Treatment on Speaking Pause Time during self-generated speech

    Evaluation of treatment group differences in change on speaking pause time from baseline to 2-months post-treatment. Metric: Pause time as percentage of total speaking time during self-generated speech (minimum=0%; maximum=100%).

    Time frame: Outcome measures will be assessed as change over a period of 13 weeks: change from baseline to 2-months Post-Treatment

  15. Treatment group differences in change from Baseline to 1-week Post-treatment on Freezing of Gate during the timed up and go task

    Evaluation of treatment group differences in change in freezing of gate from baseline to 1-week post-treatment. Metric: Timed up and go time (minimum=0; no maximum).

    Time frame: Outcome measures will be assessed as change over a period of 6 weeks: Change from baseline to 1-week Post-Treatment

  16. Treatment group differences in change from Baseline to 2-month Post-treatment on Freezing of Gate during the timed up and go task

    Evaluation of treatment group differences in change in freezing of gate from baseline to 2-month post-treatment. Metric: Timed up and go time (minimum=0; no maximum).

    Time frame: Outcome measures will be assessed as change over a period of 13 weeks: Change from baseline to 2-month Post-Treatment

06

Study locations

1 of 1 sites recruiting
  • Callier Clinical Center, The University of Texas at Dallas
    Richardson, Texas 75080, United States
    • Michael A Motes, PhD · Contact · michael.motes@utd.edu · 972-883-4846
    • John Hart, Jr, MD · Principal investigator
    • Saul A Frankford, PhD · Principal investigator
    Recruiting
07

References and documents

Individual participant data

Plan to share: Yes — De-identified individual participant data (IPD) that underlie the results reported in published articles, including baseline demographic and clinical characteristics required to replicate the findings, will be made available.

Supporting information: Study protocol, Sap

No publications or documents are linked to this record.

08

Registry details

Key details

Study ID
NCT07534397
Lead sponsor
The University of Texas at Dallas
Collaborators
University of Texas Southwestern Medical Center
Responsible party
John Hart, Jr. (Professor, The University of Texas at Dallas) — Principal investigator
First posted
Apr 16, 2026
Start date
Sep 15, 2026
Primary completion
Oct 31, 2027 (estimated)
Completion
Oct 31, 2027 (estimated)
Last update
Sep 16, 2026

Study contacts

Michael A Motes, PhD
Contact
michael.motes@utdallas.edu
972-883-4846
John Hart, MD
principal investigator · Callier Clinical Research Center, The University of Texas at Dallas
Saul A Frankford, PhD
principal investigator · Callier Clinical Research Center, The University of Texas at Dallas

Oversight

Data monitoring committee
No
FDA-regulated drug
No
FDA-regulated device
Yes
View the source record on ClinicalTrials.gov ↗

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