CClinicalTrials.gg
RecruitingNCT07238400CARDAMOMUpdated Jul 10, 2026

Cardiac Effects of Mineralocorticoid Receptor Antagonism After Preeclampsia

A Phase 2 interventional study of Eplerenone 100 mg daily and Chlorthalidone 25 mg daily in Hypertension, sponsored by Massachusetts General Hospital. Recruiting at 2 sites in United States. Open to female participants aged 18 Years to 65 Years. Per ClinicalTrials.gov, last updated 2026-07-10.

Sponsored by Massachusetts General Hospital · Phase 2, Interventional, and Treatment

Phase
Phase 2
Study type
Interventional
Enrollment
90
Allocation
Randomized
Ages
18 Years to 65 Years
Sex
Female
01

Study summary

The goal of this clinical trial is to determine if the medication eplerenone yields greater improvements in coronary microvascular function than chlorthalidone in women who experienced preeclampsia during pregnancy and subsequently developed chronic hypertension. The main Aims are:

  • To test the hypothesis that, in women with prior preeclampsia, current chronic hypertension, and concentric LV remodeling, eplerenone improves coronary microvascular function vs. chlorthalidone.
  • To test the hypothesis that, in women with prior preeclampsia, current chronic hypertension, and concentric LV remodeling, eplerenone improves cardiac structure and function vs. chlorthalidone.

Participants will:

  • First receive pre-treatment with Amlodipine for 12 weeks prior to beginning the study medication.
  • Start study treatment which involves daily self-administration of two oral capsules (eplerenone + potassium placebo or chlorthalidone + potassium), each taken once a day, for a total of 336 doses over 48 weeks.
  • Attend study visits at weeks 2, 12, 24, 36, and 48. These visits will involve collecting information, measuring blood pressure, and gathering blood and urine samples. Echocardiography (cardiac ultrasound), eye exam, and cardiac PET/CT scan will be performed during the baseline and week 48 visits.
Read the detailed description

Preeclampsia, a condition marked by hypertension and systemic endothelial/microvascular dysfunction in late pregnancy, affects 8% of childbearing U.S. women and is associated with two-fold risk of future material cardiovascular disease (CVD). The American College of Cardiology and American Heart Association now recognize preeclampsia as a sex-specific CVD risk factor to guide prescription of preventive statin therapy. Beyond this focused recommendation, however, specific strategies for CVD risk reduction in women with preeclampsia are not yet established. Recent preclinical evidence suggests that preeclampsia induces vascular smooth muscle cell mineralocorticoid receptor (MR) sensitivity that persists postpartum, promoting hypertension and CVD. Although MR signaling is known to underlie hypertension, MR activation also promotes cardiovascular, kidney, and metabolic disease via effects that are partially independent of blood pressure. Work by this team has implicated MR signaling in coronary microvascular dysfunction, a known predictor of heart failure with preserved ejection fraction (HFpEF) and CVD mortality. The central hypothesis is that, among women with prior preeclampsia who subsequently develop chronic hypertension, MR blockade will promote favorable cardiac remodeling and improve coronary microvascular function, independent of changes in blood pressure, and thereby reduce CVD risk in affected women. To test this hypothesis, the investigators propose a randomized, double-blind clinical study in humans. Women aged \<55 years with a history of preeclampsia, current chronic hypertension, and concentric left ventricular remodeling will be randomized 1:1 to receive eplerenone (mineralocorticoid receptor antagonist) or chlorthalidone (thiazide-like diuretic) with potassium supplementation for 48 weeks, targeting equivalent blood pressure control in both groups using daily home blood pressure measurement and 24-hour ambulatory blood pressure monitoring. The investigators will measure coronary microvascular function (myocardial flow reserve, i.e., hyperemic stress/rest myocardial blood flow) quantified by cardiac positron emission tomography (PET/CT) and cardiac structure and function by cardiac ultrasound at baseline and 48 weeks. It is expected that, compared with chlorthalidone, eplerenone will yield greater improvements in coronary microvascular function and myocardial diastolic function after 48 weeks of treatment. If the hypotheses are affirmed, these findings would support the targeted use of MR antagonists much earlier than recommended by current guidelines for the management of hypertension to more effectively prevent HFpEF and other CVD among women with a history of preeclampsia.

02

Conditions studied

  • Hypertension

Keywords

  • Hypertension
  • Mineralocorticoid receptor
  • chlorthalidone
  • eplerenone
  • heart
  • microvascular
  • preeclampsia
03

Who can participate

Ages eligible
18 Years to 65 Years
Sexes eligible
Female
Accepts healthy volunteers
No

Inclusion criteria

  • Female with a history of preeclampsia (defined by ACOG criteria) in a singleton pregnancy without pre-gestational chronic hypertension.
  • Current chronic hypertension (stage 1 or greater).
  • Evidence of concentric left ventricular (LV) remodeling, defined as relative LV wall thickness >0.42, with or without LV hypertrophy.
  • Age 18-65 years at time of randomization.

Exclusion criteria

Exclusion Criteria:

  • Use of a mineralocorticoid receptor antagonist (MRA) or amiloride within the past 3 months or more than 30 days within the previous 12 months.
  • Planned pregnancy, current pregnancy, or lactation.
  • Systolic BP >150 mmHg and/or diastolic BP >95 mmHg while on antihypertensives, or systolic BP >160 mmHg and/or diastolic BP >100 mmHg if untreated.
  • BMI >45 kg/m².
  • Clinical atherosclerotic cardiovascular disease, including coronary, cerebrovascular, or peripheral artery disease.
  • Diabetes mellitus.
  • LV ejection fraction \<40% or history of clinical heart failure (reduced or preserved ejection fraction).
  • Hypertrophic or other genetic cardiomyopathy.
  • Any moderate or greater valvular heart disease.
  • eGFR \<60 mL/min/1.73 m².
  • Urine microalbumin/creatinine ratio >300 mg/g at screening.
  • Abnormal electrolytes, hemoglobin, liver function tests, or TSH at screening or baseline.
  • Plasma renin activity \<1 mg/mL/hour and aldosterone >20 ng/dL (suggestive of primary aldosteronism).
  • Use of oral contraceptives, progestin depot or implant (note: progestin-containing IUD is permitted), or menopausal hormone therapy.
  • History of hypersensitivity or intolerance to calcium channel blockers, thiazides, or MRAs.
  • Active substance abuse.
  • Other serious medical illnesses or concerns about protocol adherence/mortality risk within 15 months.
  • Participation in another interventional clinical study.
  • Participants using GLP-1 receptor agonists (GLP-1RA) are eligible only if they have received continuous treatment with the same GLP-1RA agent at an unchanged maintenance dose for ≥12 months prior to enrollment. Dose changes, agent switches, or formulation changes within the 12 months preceding enrollment are not permitted. Temporary interruptions of ≤4 consecutive weeks (e.g., due to supply issues or procedural holds) are allowed, provided the same agent and dose are resumed. At the time of enrollment, there must be no planned or anticipated GLP-1RA dose escalation, dose reduction, or discontinuation. Initiation of GLP-1RA therapy after randomization is not permitted. Dose changes during follow-up are discouraged unless clinically required.
  • Use of allopurinol.
  • Use of lithium.
04

Study design

Phase
Phase 2
Primary purpose
Treatment
Allocation
Randomized
Intervention model
Parallel assignment
Masking
Quadruple (Participant, Care provider, Investigator, Outcomes assessor)
Enrollment
90 participants (estimated)

Study arms

  • Active comparator
    Eplerenone

    Participants will receive eplerenone 100 mg daily plus potassium placebo for 48 weeks

    Drug: Eplerenone 100 mg daily · Drug: Potassium Placebo

  • Active comparator
    Chlorthalidone

    Participants will receive chlorthalidone 25 mg plus potassium 20 mEq daily for 48 weeks

    Drug: Chlorthalidone 25 mg daily · Drug: Potassium Chloride

Interventions

  • DrugEplerenone 100 mg daily

    Participants with a history of preeclampsia and current chronic hypertension will receive 100mg capsules of eplerenone to self-administer daily over the 48-week duration of the study treatment.

  • DrugChlorthalidone 25 mg daily

    Participants with a history of preeclampsia with current chronic hypertension will receive 25mg capsules of chlorthalidone to self-administer daily over the 48-week duration of the study treatment.

  • DrugPotassium Placebo

    Participants taking eplerenone will also take a potassium placebo to self-administer daily over the 48-week duration of the study treatment.

  • DrugPotassium Chloride

    Participants taking chlorthalidone will also take 20 mEq of potassium to self-administer daily over the 48-week duration of the study treatment.

05

What researchers measure

Primary outcomes

  1. Change in Myocardial Flow Reserve (MFR)

    Subjects will undergo 13N cardiac PET imaging at baseline and after 48 weeks of randomized study treatment. Myocardial blood flow (MBF) will be determined during the stress and rest conditions. Myocardial flow reserve will be calculated as stress MBF divided by rest MBF.

    Time frame: Prior to randomization and after 48 weeks of randomized study treatment.

Secondary outcomes

  1. Ratio of mitral E velocity to e' [E/e']

    E/e', a measure of diastolic function, will be measured by transthoracic echocardiography.

    Time frame: Prior to randomization and after 48 weeks of randomized study treatment.

  2. Rest Myocardial Blood Flow (MBF)

    Subjects will undergo 13N cardiac PET imaging at baseline and after 48 weeks of randomized study treatment. Myocardial blood flow (MBF) will be determined during the stress and rest conditions.

    Time frame: Prior to randomization and after 48 weeks of randomized study treatment.

  3. Stress Myocardial Blood Flow (MBF)

    Subjects will undergo 13N cardiac PET imaging at baseline and after 48 weeks of randomized study treatment. Myocardial blood flow (MBF) will be determined during the stress and rest conditions.

    Time frame: Prior to randomization and after 48 weeks of randomized study treatment.

  4. Subendocardial-Specific Myocardial Flow Reserve (MFR)

    Subjects will undergo 13N cardiac PET imaging at baseline and after 48 weeks of randomized study treatment. Myocardial blood flow (MBF) will be determined during the stress and rest conditions. Subendocardial layer-specific MBF will be calculated using QPET software (Cedars-Sinai Medical Center, Los Angeles, CA). Myocardial flow reserve will be calculated as stress MBF divided by rest MBF.

    Time frame: Prior to randomization and after 48 weeks of randomized study treatment.

  5. Relative wall thickness

    Relative wall thickness is calculated as 2\*posterior wall thickness/LV end-diastolic diameter as measured by transthoracic echocardiography.

    Time frame: Prior to randomization and after 48 weeks of randomized study treatment.

  6. Early diastolic septal mitral annular velocity [septal e']

    Septal e', a measure of diastolic function, will be measured by transthoracic echocardiography using tissue Doppler.

    Time frame: Prior to randomization and after 48 weeks of randomized study treatment.

  7. Peak tricuspid regurgitant velocity

    Peak tricuspid regurgitant jet velocity, a measure of diastolic function, will be measured by transthoracic echocardiography using continuous wave Doppler.

    Time frame: Prior to randomization and after 48 weeks of randomized study treatment.

  8. Left atrial volume index

    Left atrial volume index will be measured by transthoracic echocardiogarphy using the biplane method and indexed for body surface area.

    Time frame: Prior to randomization and after 48 weeks of randomized study treatment.

  9. Left atrial reservoir strain

    Left atrial strain, a sensitive measure of end-diastolic pressure and atrial remodeling, will be quantified using TOMTEC.

    Time frame: Prior to randomization and after 48 weeks of randomized study treatment.

  10. Peak global longitudinal strain

    Left ventricular global longitudinal strain, a measure of subclinical cardiac dysfunction, will be quantified using TOMTEC.

    Time frame: Prior to randomization and after 48 weeks of randomized study treatment.

Other outcomes

  1. Renal blood flow

    Renal blood flow is calculated from 13N cardiac PET images. Rest and stress renal blood flow are estimated by fitting the tissue time-activity curves to a 2-compartment kinetic model.

    Time frame: Prior to randomization and after 48 weeks of randomized study treatment.

  2. Retinal vessel density

    For OCT ocular retinal imaging, subjects will be scanned on the commercially available spectral domain OCT machine (AngioVue OCTA, RTVue, Optovue Inc., Fremont, CA). Ocular imaging will include the 3 main retinal parameters available with the latest software package: (1) foveal avascular zone size, (2) peripapillary vessel density and internal limiting membrane-nerve fiber layer thickness, and (3) macular vessel density and macular internal limiting membrane-inner plexiform layer thickness and macular internal limiting membrane-retinal pigment epithelium full retinal thickness.

    Time frame: Prior to randomization and after 48 weeks of randomized study treatment.

  3. Concentration of High-sensitivity cardiac troponin I

    High-sensitivity cardiac troponin I, a marker of cardiovascular injury and remodeling, will be measured via blood collection.

    Time frame: Prior to randomization and after 48 weeks of randomized study treatment.

  4. Level of N-terminal pro-B-type natriuretic peptide

    NT-proBNP, a marker of cardiac wall stretch and remodeling, will be measured via blood collection.

    Time frame: Prior to randomization and after 48 weeks of randomized study treatment.

  5. High-sensitivity C-reactive protein

    We will measure high-sensitivity C-reactive protein, a marker of inflammation and cardiovascular disease risk, via blood collection.

    Time frame: Prior to randomization and after 48 weeks of randomized study treatment.

  6. Level of Interleukin-6

    IL-6, a marker of inflammation and cardiovascular disease risk, will be measured via blood collection.

    Time frame: Prior to randomization and after 48 weeks of randomized study treatment.

  7. Concentration of Transforming growth factor-beta1

    TGF-B1, a marker of fibrosis, will be measured via blood collection.

    Time frame: Prior to randomization and after 48 weeks of randomized study treatment.

  8. Concentration of procollagen type I carboxy-terminal propeptide

    PICP, a marker of fibrosis, will be measured via blood collection.

    Time frame: Prior to randomization and after 48 weeks of randomized study treatment.

  9. Urine microalbumin-to-creatinine ratio

    Microalbumin-to-creatinine will be measured through urine.

    Time frame: Prior to randomization and after 48 weeks of randomized study treatment.

  10. Score of Coronary artery calcium

    Gated CT will be performed with cardiac PET scans. Coronary artery calcium scores will be quantified using the Agatston method and can range from 0 (no plaque) to more than 1000 (extensive plaque).

    Time frame: Prior to randomization and after 48 weeks of randomized study treatment.

  11. 24-hour ambulatory blood pressure

    24-hour BP monitoring will be performed as part of the baseline and post-treatment assessments using a validated ambulatory BP monitor (WatchBP O3, microlife, Clearwater, FL). Mean 24- hour systolic and diastolic BP will be calculated from the device data.

    Time frame: Prior to randomization and after 48 weeks of randomized study treatment.

06

Study locations

2 of 2 sites recruiting
  • Brigham and Women's Hospital
    Boston, Massachusetts 02115, United States
    Recruiting
  • Massachusetts General Hospital
    Boston, Massachusetts 02115, United States
    Recruiting
07

References and documents

Individual participant data

Plan to share: No

No publications or documents are linked to this record.

08

Registry details

Key details

Study ID
NCT07238400
Lead sponsor
Massachusetts General Hospital
Collaborators
Brigham and Women's Hospital, National Heart, Lung, and Blood Institute (NHLBI)
Responsible party
Michael C. Honigberg (Clinician Investigator, Assistant Professor, Massachusetts General Hospital) — Principal investigator
First posted
Nov 20, 2025
Start date
Mar 20, 2026
Primary completion
Mar 16, 2029 (estimated)
Completion
Mar 30, 2029 (estimated)
Last update
Jul 10, 2026

Study contacts

Pearl Lee, MSc
Contact
plee23@mgh.harvard.edu
617-721-7783
Michael Honigberg, MD
principal investigator · Massachusetts General Hospital

Oversight

Data monitoring committee
Yes
FDA-regulated drug
Yes
FDA-regulated device
No
View the source record on ClinicalTrials.gov ↗

Interested in this study?

Eligibility is decided by the study team. Share this record with your doctor or contact the team directly.

Contact study team

Follow this study

Get an email when the registry record changes — status, dates, results — or when someone posts here.

Sign in to follow

Discussion

Questions and observations about this study, from anyone following it. Not medical advice, and not a channel to the study team — their contact details are on the registry record.

Sign in to join the discussion. Reading takes no account; posting does. You choose a display name, and a pseudonym is the default.

Nothing here yet. If you are running this trial, taking part in it, or weighing whether to, this is the place to say so.

Start the discussion