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CompletedNCT03402841OPINIONUpdated Aug 21, 2024Results posted

Multicentre Study of Olaparib Maintenance Monotherapy in Platinum Sensitive Relapsed Non gBRCAm Ovarian Cancer Patients

A Phase 3 interventional study of Olaparib in Non-Germline BRCA Mutated Ovarian Cancer, sponsored by AstraZeneca. Completed at 91 sites in 19 countries. Open to female participants aged 18 Years to 95 Years. Per ClinicalTrials.gov, last updated 2024-08-21.

Sponsored by AstraZeneca · Phase 3, Interventional, and Treatment

Phase
Phase 3
Study type
Interventional
Enrollment
279
Allocation
Non-randomized
Ages
18 Years to 95 Years
Sex
Female
01

Study summary

The purpose of the study is to assess the efficacy and safety of single-agent olaparib as a maintenance treatment in patients with relapsed High Grade Serous Ovarian Cancer (including patients with primary peritoneal and/or fallopian tube cancer) or high grade endometrioid cancer who do not have known deleterious or suspected deleterious germline BRCA mutations (non-gBRCAm) and who had responded following platinum based chemotherapy

Read the detailed description

Maintenance monotherapy with the potent polyadenosine 5'diphosphoribose [Poly (ADP-ribose)] polymerisation (PARP) inhibitor (PARPi) olaparib will significantly prolong progression-free survival (PFS) in platinum sensitive relapsed non-germline breast cancer susceptibility gene (BRCA) mutated ovarian cancer patients who are in complete or partial response following platinum based chemotherapy.

Olaparib is a potent PARPi (PARP-1, -2 and -3) that is being developed as an oral therapy, both as a monotherapy (including maintenance) and for combination with chemotherapy and other anticancer agents. PARP inhibition is a novel approach to targeting tumours with deficiencies in DNA repair mechanisms. PARP enzymes are essential for repairing DNA single strand breaks (SSBs). Inhibiting PARP enzymes leads to the persistence of SSBs, which are then converted to the more serious DNA double strand breaks (DSBs) during the process of DNA replication. During the process of cell division, DSBs can be efficiently repaired in normal cells by homologous recombination (HR) repair. Tumours with HR deficiencies (HRDs), such as ovarian cancers in patients with BRCA1/2 mutations, cannot accurately repair the DNA damage, which may become lethal to cells as it accumulates. In such tumour types, olaparib may offer a potentially efficacious and less toxic cancer treatment compared with currently available chemotherapy regimens.

While multiple randomized controlled trials (RCTs) have demonstrated that platinum sensitive BRCAm patients have profound response to maintenance treatment with PARP inhibitors, PARP inhibitors target cells with homologous recombination deficiency (HRD), of which BRCA mutation is only one type. Consistent with the mechanism of action of PARP inhibition, response has also been seen in multiple RCTs in patients who are platinum sensitive but whose tumours do not harbor BRCA mutations. Presumably these responders have defects in other components of HRR pathways, though currently available diagnostic technology is not adequate to reliably identify the full spectrum of HRR deficiencies. Instead, these data support the hypothesis that platinum sensitivity itself is a clinical selection factor for HRD.

02

Conditions studied

  • Non-Germline BRCA Mutated Ovarian Cancer

Keywords

  • Ovarian cancer
  • non-gBRCA
  • Platinum
  • Chemotherapy
03

Who can participate

Ages eligible
18 Years to 95 Years
Sexes eligible
Female
Accepts healthy volunteers
No

Eligibility criteria

Key Inclusion Criteria:

  • Female patients with histologically diagnosed relapsed HGSOC (including primary peritoneal and / or fallopian tube cancer) or high grade endometrioid ovarian cancer
  • Documented gBRCA1/2 mutation status
  • Patients must have completed at least 2 previous courses of platinum containing therapy
  • Patients must have normal organ and bone marrow function measured within 28 days of starting study treatment
  • ECOG performance status 0-1 (see Appendix E)
  • Patients must have a life expectancy ≥16 weeks
  • Postmenopausal or evidence of non-childbearing status for women of childbearing potential: negative urine or serum pregnancy test within 28 days of study treatment and confirmed prior to treatment on day 1
  • At least one lesion (measurable and/or non-measurable) that can be accurately assessed at baseline with computed tomography (CT) or magnetic resonance imaging (MRI) and is suitable for repeated assessment OR No evidence of disease following a complete response to chemotherapy
  • An appropriately prepared tumour sample from the cancer, of sufficient quantity and quality (as specified in the Central Laboratory Services Manual) must be available for future central testing of tumour genetic status

Exclusion Criteria:

  • Patients receiving any systemic hormonal therapy, chemotherapy or radiotherapy (except for palliative reasons) within 3 weeks prior to start of study treatment
  • Any previous treatment with PARP inhibitor, including olaparib
  • Patients with a germline BRCA mutation that is predicted to be deleterious or suspected deleterious (known or predicted to be detrimental / lead to loss of function)
  • Other malignancy unless curatively treated with no evidence of disease for ≥5 years except: adequately treated non-melanoma skin cancer, curatively treated in situ cancer of the cervix, ductal carcinoma in situ (DCIS), Stage 1, grade 1 endometrial carcinoma.
  • Concomitant use of known strong CYP3A inhibitors and strong (or moderate CYP3A inducers
  • Persistent toxicities (≥ Grade 2 Common Terminology Criteria for Adverse Event (CTCAE) adverse event) caused by previous cancer therapy, excluding alopecia
  • Patients with myelodysplastic syndrome (MDS)/acute myeloid leukaemia (AML) or with features suggestive of MDS/AML
  • Patients with symptomatic uncontrolled brain metastases
04

Study design

Phase
Phase 3
Primary purpose
Treatment
Allocation
Non-randomized
Intervention model
Single group
Masking
None (open label)
Enrollment
279 participants (actual)

Study arms

  • Experimental
    Olaparib

    Olaparib will be supplied as film-coated tablets containing 150 mg or 100 mg of olaparib. Patients will be administered olaparib orally twice daily (bid) at 300 mg.

    Drug: Olaparib

Interventions

  • DrugOlaparib

    300 mg twice daily - oral

    Also known as: Lynparza

05

What researchers measure

Primary outcomes

  1. Progression Free Survival (PFS)

    PFS is defined as the time from date of first dose until the date of objective radiological disease progression. Assessed according to modified Response Evaluation Criteria In Solid Tumours Version 1.1 (RECIST 1.1) or death (by any cause in the absence of progression). Progression was determined by investigator assessment, RECIST 1.1. Calculated using the Kaplan-Meier technique. Confidence intervals (CI) for median PFS was derived based on Brookmeyer-Crowley method.

    Time frame: Up to maximum of 32 months

Secondary outcomes

  1. Time to First Subsequent Therapy or Death (TFST)

    TFST is defined as the time from date of first dose to date of first subsequent treatment commencement or death due to any cause if this occurs before commencement of first subsequent treatment. Calculated using the Kaplan-Meier technique. CI for median TFST was derived based on Brookmeyer-Crowley method.

    Time frame: Up to a maximum of 43 months

  2. Time to Treatment Discontinuation or Death (TDT)

    TDT is defined as the time from date of first dose to date of study drug discontinuation or death due to any cause if this occurs before study drug discontinuation. Calculated using the Kaplan-Meier technique. CI for median TDT was derived based on Brookmeyer-Crowley method.

    Time frame: Up to a maximum of 43 months

  3. PFS by Homologous Recombination Deficiency (HRD)/ Breast Cancer Susceptibility Gene Mutation (Mutated) (BRCAm) Status

    HRD/BRCAm status was based on the central blood and tumour assessments. Assessed according to modified RECIST 1.1 or death (by any cause in the absence of progression). Progression was determined by investigator assessment, RECIST 1.1. Calculated using the Kaplan-Meier technique. CI for median PFS was derived based on Brookmeyer-Crowley method.

    Time frame: Up to maximum of 32 months

  4. Chemotherapy-free Interval (CT-FI)

    CT-FI is defined as the time from the date of the last dose of platinum chemotherapy prior to olaparib maintenance therapy until the date of initiation of the next anticancer therapy. Calculated using the Kaplan-Meier technique. CI for median CT-FI was derived based on Brookmeyer-Crowley method.

    Time frame: Up to a maximum of 43 months

  5. Overall Survival (OS)

    OS is defined as the time from the date of first dose of olaparib to the date of death from any cause. Calculated using the Kaplan-Meier technique. CI for median OS was derived based on Brookmeyer-Crowley method.

    Time frame: Up to a maximum of 43 months

  6. Percentage of Patients With Any Improvement From Baseline in Trial Outcome Index (TOI) Score at Any Point During the Treatment Period

    Improvement was defined as a functional assessment of cancer therapy - ovarian (FACT-O) TOI response of "any improvement" at any time point over the course of treatment. The TOI is an established single targeted index composed of the following scales of the FACT-O: physical and functional well-being and additional concerns. The range of possible scores for the FACT-O TOI is 0-100, with a higher score indicating better health related quality of life (HRQoL). An increase in score from baseline indicates an improvement in HRQoL.

    Time frame: Baseline up to a maximum of 32 months

  7. Percentage of Patients With a 10-Point Deterioration From Baseline in TOI Score at Any Point During the Treatment Period

    10-point deterioration was defined as a FACT-O TOI response of "10-point deterioration" at any time point over the course of treatment. The TOI is an established single targeted index composed of the following scales of the FACT-O: physical and functional well-being and additional concerns. The range of possible scores for the FACT-O TOI is 0-100, with a higher score indicating better HRQoL. A decrease in score of at least 10 points from baseline was defined as a clinically meaningful deterioration.

    Time frame: Baseline up to a maximum of 32 months

06

Results

Posted Oct 11, 2021

Participant flow

This was a Phase IIIb, single-arm, open-label multicentre study to assess the efficacy and safety of single-agent olaparib as a maintenance treatment in eligible patients. A total of 279 patients from 17 countries were enrolled in this study.

Participant flow — Overall Study
MilestoneOlaparib
Started279
Completed128
Not completed151
Withdrew: Withdrawal by subject3
Withdrew: Lost to follow-up2
Withdrew: Death146

Outcome measures

PrimaryProgression Free Survival (PFS)

PFS is defined as the time from date of first dose until the date of objective radiological disease progression. Assessed according to modified Response Evaluation Criteria In Solid Tumours Version 1.1 (RECIST 1.1) or death (by any cause in the absence of progression). Progression was determined by investigator assessment, RECIST 1.1. Calculated using the Kaplan-Meier technique. Confidence intervals (CI) for median PFS was derived based on Brookmeyer-Crowley method.

Time frame:
Up to maximum of 32 months
Reported as:
Median · months
Progression Free Survival (PFS)
monthsOlaparib
Progression Free Survival (PFS)9.2 (7.6 to 10.9)
SecondaryTime to First Subsequent Therapy or Death (TFST)

TFST is defined as the time from date of first dose to date of first subsequent treatment commencement or death due to any cause if this occurs before commencement of first subsequent treatment. Calculated using the Kaplan-Meier technique. CI for median TFST was derived based on Brookmeyer-Crowley method.

Time frame:
Up to a maximum of 43 months
Reported as:
Median · months
Time to First Subsequent Therapy or Death (TFST)
monthsOlaparib
Time to First Subsequent Therapy or Death (TFST)13.9 (11.5 to 16.6)
SecondaryTime to Treatment Discontinuation or Death (TDT)

TDT is defined as the time from date of first dose to date of study drug discontinuation or death due to any cause if this occurs before study drug discontinuation. Calculated using the Kaplan-Meier technique. CI for median TDT was derived based on Brookmeyer-Crowley method.

Time frame:
Up to a maximum of 43 months
Reported as:
Median · months
Time to Treatment Discontinuation or Death (TDT)
monthsOlaparib
Time to Treatment Discontinuation or Death (TDT)9.6 (7.8 to 11.1)
SecondaryPFS by Homologous Recombination Deficiency (HRD)/ Breast Cancer Susceptibility Gene Mutation (Mutated) (BRCAm) Status

HRD/BRCAm status was based on the central blood and tumour assessments. Assessed according to modified RECIST 1.1 or death (by any cause in the absence of progression). Progression was determined by investigator assessment, RECIST 1.1. Calculated using the Kaplan-Meier technique. CI for median PFS was derived based on Brookmeyer-Crowley method.

Time frame:
Up to maximum of 32 months
Reported as:
Median · months
PFS by Homologous Recombination Deficiency (HRD)/ Breast Cancer Susceptibility Gene Mutation (Mutated) (BRCAm) Status
monthsOlaparib
HRD status positive and/or sBRCAm subgroup11.1 (9.2 to 14.6)
HRD status positive, non-BRCAm subgroup9.7 (8.1 to 13.6)
HRD status negative subgroup7.3 (5.5 to 9.0)
sBRCAm subgroup16.4 (12.8 to NA)
gBRCAm subgroup12.1 (3.7 to NA)
SecondaryChemotherapy-free Interval (CT-FI)

CT-FI is defined as the time from the date of the last dose of platinum chemotherapy prior to olaparib maintenance therapy until the date of initiation of the next anticancer therapy. Calculated using the Kaplan-Meier technique. CI for median CT-FI was derived based on Brookmeyer-Crowley method.

Time frame:
Up to a maximum of 43 months
Reported as:
Median · months
Chemotherapy-free Interval (CT-FI)
monthsOlaparib
Chemotherapy-free Interval (CT-FI)17.9 (13.8 to 23.3)
SecondaryOverall Survival (OS)

OS is defined as the time from the date of first dose of olaparib to the date of death from any cause. Calculated using the Kaplan-Meier technique. CI for median OS was derived based on Brookmeyer-Crowley method.

Time frame:
Up to a maximum of 43 months
Reported as:
Median · months
Overall Survival (OS)
monthsOlaparib
Overall Survival (OS)32.7 (29.5 to 35.3)
SecondaryPercentage of Patients With Any Improvement From Baseline in Trial Outcome Index (TOI) Score at Any Point During the Treatment Period

Improvement was defined as a functional assessment of cancer therapy - ovarian (FACT-O) TOI response of "any improvement" at any time point over the course of treatment. The TOI is an established single targeted index composed of the following scales of the FACT-O: physical and functional well-being and additional concerns. The range of possible scores for the FACT-O TOI is 0-100, with a higher score indicating better health related quality of life (HRQoL). An increase in score from baseline indicates an improvement in HRQoL.

Time frame:
Baseline up to a maximum of 32 months
Reported as:
Number · percentage of patients
Percentage of Patients With Any Improvement From Baseline in Trial Outcome Index (TOI) Score at Any Point During the Treatment Period
percentage of patientsOlaparib
Percentage of Patients With Any Improvement From Baseline in Trial Outcome Index (TOI) Score at Any Point During the Treatment Period64.3 (58.0 to 70.2)
SecondaryPercentage of Patients With a 10-Point Deterioration From Baseline in TOI Score at Any Point During the Treatment Period

10-point deterioration was defined as a FACT-O TOI response of "10-point deterioration" at any time point over the course of treatment. The TOI is an established single targeted index composed of the following scales of the FACT-O: physical and functional well-being and additional concerns. The range of possible scores for the FACT-O TOI is 0-100, with a higher score indicating better HRQoL. A decrease in score of at least 10 points from baseline was defined as a clinically meaningful deterioration.

Time frame:
Baseline up to a maximum of 32 months
Reported as:
Number · percentage of patients
Percentage of Patients With a 10-Point Deterioration From Baseline in TOI Score at Any Point During the Treatment Period
percentage of patientsOlaparib
Percentage of Patients With a 10-Point Deterioration From Baseline in TOI Score at Any Point During the Treatment Period42.6 (36.3 to 49.0)

Adverse events

Collected over Includes adverse events with an onset date on or after the date of first dose and up to and including 30 days following the date of last dose of olaparib, up to a maximum of 43 months. Non-serious events are listed at a 0% frequency threshold.

Adverse event summary by group
GroupDeathsSeriousOther
Olaparib 300mg BID146/279 (52.3%)58/279 (20.8%)268/279 (96.1%)
Most frequent serious events
Showing 10 of 51
Most frequent serious events
EventOlaparib 300mg BID
AnaemiaBlood and lymphatic system disorders22/279
PneumoniaInfections and infestations3/279
Pulmonary embolismRespiratory, thoracic and mediastinal disorders3/279
Intestinal obstructionGastrointestinal disorders3/279
Covid-19Infections and infestations2/279
SepsisInfections and infestations2/279
Breast cancerNeoplasms benign, malignant and unspecified (incl cysts and polyps)2/279
Small intestinal obstructionGastrointestinal disorders2/279
ThrombocytopeniaBlood and lymphatic system disorders1/279
Biliary dyskinesiaHepatobiliary disorders1/279
Most frequent other events
Showing 10 of 354
Most frequent other events
EventOlaparib 300mg BID
NauseaGastrointestinal disorders136/279
AnaemiaBlood and lymphatic system disorders102/279
FatigueGeneral disorders82/279
AstheniaGeneral disorders46/279
VomitingGastrointestinal disorders45/279
Abdominal painGastrointestinal disorders43/279
DysgeusiaNervous system disorders40/279
DiarrhoeaGastrointestinal disorders40/279
Decreased appetiteMetabolism and nutrition disorders32/279
CoughRespiratory, thoracic and mediastinal disorders31/279

Baseline characteristics

The Full Analysis Set (FAS) included all enrolled patients assigned to olaparib.

Age, Continuous
Age, Continuous(years)Olaparib
Mean64.0 ± 9.19
Age, Customized
Age, Customized(Participants)Olaparib
<50 years22
>=50 - <65 years110
>=65 - <75 years113
>=75 years34
Sex: Female, Male
Sex: Female, Male(Participants)Olaparib
Female279
Male0
Race/Ethnicity, Customized
Race/Ethnicity, Customized(Participants)Olaparib
White273
Black or African American1
Asian2
Unspecified2
Missing1
Race/Ethnicity, Customized
Race/Ethnicity, Customized(Participants)Olaparib
Hispanic or Latino7
Not Hispanic or Latino271
Missing1
07

Study locations

91 sites
  • Research Site
    Graz, 8036, Austria
  • Research Site
    Innsbruck, 6020, Austria
  • Research Site
    Vienna, 1090, Austria
  • Research Site
    Wein, 1130, Austria
  • Research Site
    Bruxelles, 1200, Belgium
  • Research Site
    Leuven, 3000, Belgium
  • Research Site
    Loverval, 6280, Belgium
  • Research Site
    Namur, 5000, Belgium
  • Research Site
    Wilrijk, 2610, Belgium
  • Research Site
    Plovdiv, 4000, Bulgaria
  • Research Site
    Plovdiv, 4004, Bulgaria
  • Research Site
    Sofia, 1330, Bulgaria
  • Research Site
    Victoria, British Columbia V8R 6V5, Canada
  • Research Site
    London, Ontario N6A 5W9, Canada
  • Research Site
    Ottawa, Ontario K1H 8L6, Canada
  • Research Site
    Toronto, Ontario M5G2M9, Canada
  • Research Site
    Montreal, Quebec H2X 0A9, Canada
  • Research Site
    Sherbrooke, Quebec J1H 5N4, Canada
  • Research Site
    Brno, 625 00, Czechia
  • Research Site
    Novy Jicin, 74101, Czechia
  • Research Site
    Olomouc, 775 20, Czechia
  • Research Site
    Ostrava, 708 52, Czechia
  • Research Site
    Praha 2, 128 08, Czechia
  • Research Site
    Arhus, 8200, Denmark
  • Research Site
    Ålborg, 9100, Denmark
  • Research Site
    Kuopio, 70210, Finland
  • Research Site
    Oulu, 90029, Finland
  • Research Site
    Tampere, 33520, Finland
  • Research Site
    Afula, 18101, Israel
  • Research Site
    Beer Sheva, Israel
  • Research Site
    Haifa, 91096, Israel
  • Research Site
    Jerusalem, 91031, Israel
  • Research Site
    Kfar Saba, 4428164, Israel
  • Research Site
    Ramat Gan, 52621, Israel
  • Research Site
    Ancona, 60020, Italy
  • Research Site
    Brescia, 25123, Italy
  • Research Site
    Lecco, 23900, Italy
  • Research Site
    Milano, 20141, Italy
  • Research Site
    Padova, 35128, Italy
  • Research Site
    Torino, 10126, Italy
  • Research Site
    Torino, 10128, Italy
  • Research Site
    Breda, 4818 CK, Netherlands
  • Research Site
    Tilburg, 5022 GC, Netherlands
  • Research Site
    Bergen, 5053, Norway
  • Research Site
    Oslo, 424, Norway
  • Research Site
    Gdynia, 81-519, Poland
  • Research Site
    Kraków, 31-501, Poland
  • Research Site
    Lublin, 20-090, Poland
  • Research Site
    Olsztyn, 10-513, Poland
  • Research Site
    Poznań, 60-569, Poland
  • Research Site
    Warszawa, 02-781, Poland
  • Research Site
    Warszawa, 04-141, Poland
  • Research Site
    Coimbra, 3000-073, Portugal
  • Research Site
    Lisboa, 1400-048, Portugal
  • Research Site
    Bucuresti, 031422, Romania
  • Research Site
    Cluj Napoca, 400058, Romania
  • Research Site
    Cluj-Napoca, 400015, Romania
  • Research Site
    Ljubljana, 1000, Slovenia
  • Research Site
    Badalona, 08916, Spain
  • Research Site
    Barcelona, 08036, Spain
  • Research Site
    Barcelona, 08041, Spain
  • Research Site
    Barcelona, 8035, Spain
  • Research Site
    Córdoba, 14004, Spain
  • Research Site
    Girona, 17007, Spain
  • Research Site
    Hospitalet deLlobregat, 08907, Spain
  • Research Site
    Madrid, 28027, Spain
  • Research Site
    Madrid, 28046, Spain
  • Research Site
    Málaga, 29011, Spain
  • Research Site
    Pamplona, 31008, Spain
  • Research Site
    Sevilla, 41009, Spain
  • Research Site
    Valencia, 46009, Spain
  • Research Site
    Valencia, 46026, Spain
  • Research Site
    Linköping, 581 85, Sweden
  • Research Site
    Lund, 221 85, Sweden
  • Research Site
    Basel, 4031, Switzerland
  • Research Site
    Bellinzona, 6500, Switzerland
  • Research Site
    Bern, 3010, Switzerland
  • Research Site
    Frauenfeld, 8501, Switzerland
  • Research Site
    Genève 14, 1205, Switzerland
  • Research Site
    Lausanne, 1011, Switzerland
  • Research Site
    Zürich, 8091, Switzerland
  • Research Site
    Aberdeen, AB25 2ZN, United Kingdom
  • Research Site
    Colchester, CO4 5JL, United Kingdom
  • Research Site
    Exeter, EX2 5DW, United Kingdom
  • Research Site
    Leeds, LS9 7TF, United Kingdom
  • Research Site
    Leicester, LE1 5WW, United Kingdom
  • Research Site
    London, SE1 9RT, United Kingdom
  • Research Site
    London, WC1N 3BG, United Kingdom
  • Research Site
    Manchester, M20 4BX, United Kingdom
  • Research Site
    Northampton, NN1 5BD, United Kingdom
  • Research Site
    Scarborough, YO12 6QL, United Kingdom
08

References and documents

Publications

  • Fong PC, Boss DS, Yap TA, Tutt A, Wu P, Mergui-Roelvink M, Mortimer P, Swaisland H, Lau A, O'Connor MJ, Ashworth A, Carmichael J, Kaye SB, Schellens JH, de Bono JS. Inhibition of poly(ADP-ribose) polymerase in tumors from BRCA mutation carriers. N Engl J Med. 2009 Jul 9;361(2):123-34. doi: 10.1056/NEJMoa0900212. Epub 2009 Jun 24. PubMed 19553641 ↗
  • Rottenberg S, Jaspers JE, Kersbergen A, van der Burg E, Nygren AO, Zander SA, Derksen PW, de Bruin M, Zevenhoven J, Lau A, Boulter R, Cranston A, O'Connor MJ, Martin NM, Borst P, Jonkers J. High sensitivity of BRCA1-deficient mammary tumors to the PARP inhibitor AZD2281 alone and in combination with platinum drugs. Proc Natl Acad Sci U S A. 2008 Nov 4;105(44):17079-84. doi: 10.1073/pnas.0806092105. Epub 2008 Oct 29. PubMed 18971340 ↗
  • Murai J, Huang SY, Das BB, Renaud A, Zhang Y, Doroshow JH, Ji J, Takeda S, Pommier Y. Trapping of PARP1 and PARP2 by Clinical PARP Inhibitors. Cancer Res. 2012 Nov 1;72(21):5588-99. doi: 10.1158/0008-5472.CAN-12-2753. PubMed 23118055 ↗
  • Mirza MR, Monk BJ, Herrstedt J, Oza AM, Mahner S, Redondo A, Fabbro M, Ledermann JA, Lorusso D, Vergote I, Ben-Baruch NE, Marth C, Madry R, Christensen RD, Berek JS, Dorum A, Tinker AV, du Bois A, Gonzalez-Martin A, Follana P, Benigno B, Rosenberg P, Gilbert L, Rimel BJ, Buscema J, Balser JP, Agarwal S, Matulonis UA; ENGOT-OV16/NOVA Investigators. Niraparib Maintenance Therapy in Platinum-Sensitive, Recurrent Ovarian Cancer. N Engl J Med. 2016 Dec 1;375(22):2154-2164. doi: 10.1056/NEJMoa1611310. Epub 2016 Oct 7. PubMed 27717299 ↗
  • Helleday T. The underlying mechanism for the PARP and BRCA synthetic lethality: clearing up the misunderstandings. Mol Oncol. 2011 Aug;5(4):387-93. doi: 10.1016/j.molonc.2011.07.001. Epub 2011 Jul 22. PubMed 21821475 ↗
  • Hay T, Matthews JR, Pietzka L, Lau A, Cranston A, Nygren AO, Douglas-Jones A, Smith GC, Martin NM, O'Connor M, Clarke AR. Poly(ADP-ribose) polymerase-1 inhibitor treatment regresses autochthonous Brca2/p53-mutant mammary tumors in vivo and delays tumor relapse in combination with carboplatin. Cancer Res. 2009 May 1;69(9):3850-5. doi: 10.1158/0008-5472.CAN-08-2388. Epub 2009 Apr 21. PubMed 19383921 ↗
  • Ledermann J, Harter P, Gourley C, Friedlander M, Vergote I, Rustin G, Scott C, Meier W, Shapira-Frommer R, Safra T, Matei D, Macpherson E, Watkins C, Carmichael J, Matulonis U. Olaparib maintenance therapy in platinum-sensitive relapsed ovarian cancer. N Engl J Med. 2012 Apr 12;366(15):1382-92. doi: 10.1056/NEJMoa1105535. Epub 2012 Mar 27. PubMed 22452356 ↗
  • Pujade-Lauraine E, Ledermann JA, Selle F, Gebski V, Penson RT, Oza AM, Korach J, Huzarski T, Poveda A, Pignata S, Friedlander M, Colombo N, Harter P, Fujiwara K, Ray-Coquard I, Banerjee S, Liu J, Lowe ES, Bloomfield R, Pautier P; SOLO2/ENGOT-Ov21 investigators. Olaparib tablets as maintenance therapy in patients with platinum-sensitive, relapsed ovarian cancer and a BRCA1/2 mutation (SOLO2/ENGOT-Ov21): a double-blind, randomised, placebo-controlled, phase 3 trial. Lancet Oncol. 2017 Sep;18(9):1274-1284. doi: 10.1016/S1470-2045(17)30469-2. Epub 2017 Jul 25. Erratum In: Lancet Oncol. 2017 Sep;18(9):e510. doi: 10.1016/S1470-2045(17)30639-3. PubMed 28754483 ↗
  • Poveda A, Lheureux S, Colombo N, Cibula D, Lindemann K, Weberpals J, Bjurberg M, Oaknin A, Sikorska M, Gonzalez-Martin A, Madry R, Perez MJR, Ledermann J, Davidson R, Blakeley C, Bennett J, Barnicle A, Skof E. Olaparib maintenance monotherapy in platinum-sensitive relapsed ovarian cancer patients without a germline BRCA1/BRCA2 mutation: OPINION primary analysis. Gynecol Oncol. 2022 Mar;164(3):498-504. doi: 10.1016/j.ygyno.2021.12.025. Epub 2022 Jan 19. PubMed 35063276 ↗
  • Poveda AM, Davidson R, Blakeley C, Milner A. Olaparib maintenance monotherapy in platinum-sensitive, relapsed ovarian cancer without germline BRCA mutations: OPINION Phase IIIb study design. Future Oncol. 2019 Nov;15(32):3651-3663. doi: 10.2217/fon-2019-0343. Epub 2019 Sep 25. PubMed 31553234 ↗

Study documents

  • Study protocol · Oct 26, 2018
  • Statistical analysis plan · Dec 9, 2019

Documents are hosted by the registry — open the source record to download them.

Individual participant data

Plan to share: Yes — Qualified researchers can request access to anonymized individual patient-level data from AstraZeneca group of companies sponsored clinical trials via the request portal. All request will be evaluated as per the AZ disclosure commitment: https://astrazenecagrouptrials.pharmacm.com/ST/Submission/Disclosure. Yes, indicates that AZ are accepting requests for IPD, but this does not mean all requests will be shared.

Supporting information: Study protocol, Sap

09

Registry details

Key details

Study ID
NCT03402841
Lead sponsor
AstraZeneca
Collaborators
Iqvia Pty Ltd, Myriad Genetics, Inc., Covance, Theradex, Parexel
Responsible party
Sponsor
First posted
Jan 18, 2018
Start date
Jan 30, 2018
Primary completion
Oct 2, 2020
Completion
Mar 10, 2022
Results posted
Oct 11, 2021
Last update
Aug 21, 2024

Study contacts

Chris Wilks
study director · AstraZeneca

Oversight

Data monitoring committee
No
FDA-regulated drug
No
FDA-regulated device
No
View the source record on ClinicalTrials.gov ↗

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