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RecruitingNCT07552636DiAcMoUpdated Aug 31, 2026

Disease Activity Monitoring in Patients With Giant Cell Arteritis Study

An observational study in Giant Cell Arteritis (GCA), sponsored by University of Aarhus. Recruiting at 7 sites in Denmark. Open to participants aged 50 Years and older. Per ClinicalTrials.gov, last updated 2026-08-31.

Sponsored by University of Aarhus · Observational

Study type
Observational
Model
Cohort
Time perspective
Prospective
Enrollment
175
Ages
50 Years and older
Sex
All
01

Study summary

Giant Cell Arteritis (GCA) is a vasculitis of medium- and large-sized arteries in older adults that may lead to serious vascular complications, including permanent vision loss and aortic aneurysm formation. Glucocorticoids are effective, but relapse during tapering is common and poses a major clinical challenge, potentially contributing to prolonged glucocorticoid exposure. Symptoms are often nonspecific and conventional inflammatory markers lack sufficient reliability, particularly in patients treated with drugs targeting the interleukin-6 pathway. Thus, this project aims to evaluate different tools assisting disease activity monitoring and/or predict future relapses and higher treatment requirements. Up to 175 patients with GCA in remission will be enrolled to ensure that 144 participants complete 1 year of follow-up. Participants undergo vascular ultrasonography, including double-blinded assessment at suspected relapse, complete patient-reported outcome measures, and provide biobank blood samples.

02

Conditions studied

  • Giant Cell Arteritis (GCA)

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Keywords

  • vascular ultrasonography
  • disease activity monitoring
  • giant cell arteritis
  • patient-reported outcome measures
03

Who can participate

Ages eligible
50 Years and older
Sexes eligible
All
Accepts healthy volunteers
No
Sampling method
Non-probability sample

Study population

Patients with giant cell arteritis (ICD-10: M31.5, M31.6) in clinical remission but still undergoing glucocorticoid tapering or tapering of glucocorticoid-sparing treatment.

Inclusion criteria

  1. Clinical GCA diagnosis established/confirmed by a rheumatologist and positive GCA imaging or biopsy at diagnosis \< 3 years.
  2. Clinical remission at the time of inclusion, defined as

    • Having adhered for ≥ 8 weeks prior to inclusion to either (a) the planned tapering of glucocorticoid therapy, or (b) the planned glucocorticoid-sparing DMARD treatment (with or without glucocorticoids).
    • Absence of, or no worsening of, symptoms attributed to GCA in ≥ 8 weeks.
    • Normal CRP level (\< 10 mg/L) within 7 days before inclusion.
  3. Current prednisolone dosage of ≥ 5 mg if glucocorticoid monotherapy.
  4. A continuous tapering of monotherapy or combination therapy is planned.
  5. Age > 50 years.
  6. Study participants must be able to speak and understand spoken and written Danish.

Exclusion criteria

Exclusion Criteria:

  1. Intra-articular, intravenous or intramuscular glucocorticoid ≤ 7 weeks prior to inclusion.
  2. Study participants who are unable to complete online questionnaires cannot participate in the GCA-PRO component of the study.
  3. Presence of cognitive impairment, including clinically significant dementia, that may interfere with the ability to provide informed consent or comply with study procedures.
04

Study design

Observational model
Cohort
Time perspective
Prospective
Enrollment
175 participants (estimated)
Patient registry
No
Biospecimen retention
Samples without dna
05

What researchers measure

Primary outcomes

  1. Sensitivity and specificity of change in OGUS from inclusion to suspected relapse

    Sensitivity and specificity of change in OMERACT Ultrasonography GCA Score (OGUS) from inclusion to suspected relapse (Follow-up 1a) for relapse/non-relapse using the reassessment at Follow-up 2 as the reference standard.

    Time frame: Within 10 months

Secondary outcomes

  1. Proportion of correctly classified relapse/non-relapse by vascular ultrasonography among participants with clinically uncertain relapse

    Proportion of correctly classified relapse/non-relapse by vascular ultrasonography among participants with clinically uncertain relapse (Follow-up 1a) using the reassessment at Follow-up 2 as the reference standard. Key secondary.

    Time frame: Within 10 months

  2. Predictive cut-off values of vascular ultrasonography scores at remission for relapse

    The predictive cut-off values of vascular ultrasonography scores (OGUS and halo count) at remission (inclusion) for relapse within 12 months of inclusion using Follow-up 2 as the reference standard. Key secondary.

    Time frame: 12 months

  3. Change in GCA-PRO scores from remission to relapse

    Change in Giant Cell Arteritis Patient Reported Outcome Measure (GCA-PRO) scores from remission (inclusion) to relapse (Follow-up 1a) using the reassessment at Follow-up 2 as reference standard. The total score for the GCA-PRO ranges from 0 to 90, where 0 indicates no impact on health-related quality of life (HRQoL) and 90 indicates high disease impact (poor HRQoL). Key secondary.

    Time frame: Within 10 months

  4. Sensitivity and specificity of change in halo count from inclusion to suspected relapse

    The sensitivity and specificity of change in halo count from remission (inclusion) to Follow-up 1a for relapse/non-relapse using the reassessment at Follow-up 2 as the reference standard.

    Time frame: Within 10 months

  5. Sensitivity and specificity of vascular ultrasonography scores at suspected relapse

    The sensitivity and specificity of US scores (OMERACT Ultrasonography GCA Score and halo count) at suspected relapse (Follow-up 1) for relapse/non-relapse using the reassessment at Follow-up 2 as the reference standard.

    Time frame: Within 10 months

  6. Sensitivity and specificity of vascular ultrasonography scores for relapse/non-relapse in subgroups defined by OMERACT ultrasonography morphology

    The sensitivity and specificity of vascular ultrasonography scores for relapse/non-relapse in subgroups defined by OMERACT US morphology (normal, active vasculitis, chronic vasculitis, atherosclerotic) using the reassessment at Follow-up 2 as the reference standard.

    Time frame: Within 10 months

  7. Change in probability of relapse and non-relapse before and after vascular ultrasonography

    Change in probability of relapse and non-relapse before and after vascular ultrasonography using clinician-rated pre-test probability and ultrasonography likelihood ratios.

    Time frame: Within 10 months

  8. Time from suspected relapse to clinical conclusion in participants with clinically uncertain relapse that were correctly classified with vascular ultrasonography

    The time (potentially saved) from suspected relapse (Follow-up 1a) to clinical conclusion (relapse review) in participants with clinically uncertain relapse that were correctly classified with vascular ultrasonography compared to the reference standard (Follow-up 2).

    Time frame: 12 months

  9. Number of supplementary tests ordered at suspected relapse to clinical conclusion in participants with clinically uncertain relapses/non-relapse that were correctly classified by vascular ultrasonography

    The number of supplementary tests ordered at suspected relapse (Follow-up 1a) to clinical conclusion (relapse review) in participants with clinically uncertain relapses/non-relapse that were correctly classified by vascular ultrasonography using the reassessment at Follow-up 2 as the reference standard.

    Time frame: 12 months

  10. Time to relapse over 12 months in relation to vascular ultrasonography scores at inclusion

    Time to relapse over 12 months evaluated in relation to vascular ultrasonography scores at inclusion (remission).

    Time frame: 12 months

  11. Change in GCA-PRO scores at relapse and 10 days after treatment escalation.

    Change in GCA-PRO scores at relapse and 10 days after treatment escalation. The total score for the GCA-PRO ranges from 0 to 90, where 0 indicates no impact on HRQoL and 90 indicates high disease impact (poor HRQoL).

    Time frame: Within 10 months

  12. Convergence of GCA-PRO scores with other surrogate markers of disease activity from remission to relapse

    Convergence of GCA-PRO scores with, respectively, c-reactive protein levels, patient and physician global activity numeric rating scale scores, and vascular ultrasonography scores (halo count and OGUS) from remission (inclusion) to relapse (Follow-up 1a). The total score for the GCA-PRO ranges from 0 to 90, where 0 indicates no impact on HRQoL and 90 indicates high disease impact (poor HRQoL).

    Time frame: Within 10 months

  13. Sensitivity and specificity of change in GCA-PRO scores from inclusion to suspected relapse

    The sensitivity and specificity of change in GCA-PRO scores from inclusion to suspected relapse (Follow-up 1a) for relapse/non-relapse using the reassessment at Follow-up 2 as the reference standard. The total score for the GCA-PRO ranges from 0 to 90, where 0 indicates no impact on HRQoL and 90 indicates high disease impact (poor HRQoL).

    Time frame: Within 10 months

  14. Sensitivity and specificity of GCA-PRO scores at suspected relapse

    The sensitivity and specificity of GCA-PRO scores at suspected relapse (Follow-up 1a) for relapse/non-relapse using the reassessment at Follow-up 2 as the reference standard. The total score for the GCA-PRO ranges from 0 to 90, where 0 indicates no impact on HRQoL and 90 indicates high disease impact (poor HRQoL).

    Time frame: Within 10 months

  15. Relapse within 12 months evaluated in relation to GCA-PRO scores at inclusion.

    Relapse within 12 months evaluated in relation to GCA-PRO scores at inclusion. The total score for the GCA-PRO ranges from 0 to 90, where 0 indicates no impact on HRQoL and 90 indicates high disease impact (poor HRQoL).

    Time frame: 12 months

  16. Time to relapse over 12 months evaluated in relation to GCA-PRO scores at inclusion.

    Time to relapse over 12 months evaluated in relation to GCA-PRO scores at inclusion. The total score for the GCA-PRO ranges from 0 to 90, where 0 indicates no impact on HRQoL and 90 indicates high disease impact (poor HRQoL).

    Time frame: 12 months

06

Study locations

4 of 7 sites recruiting
  • Aalborg University Hospital, Department of Rheumatology
    Aalborg, 9000, Denmark
    • Salome Kristensen, MD, PhD · Contact · sakr@rn.dk · +45 97664015
    Not yet recruiting
  • Aarhus University Hospital, Department of Rheumatology
    Aarhus, 8200, Denmark
    Recruiting
  • Rigshospitalet Glostrup, Center for Rheumatology and Spine Diseases Rigshospitalet
    Glostrup Municipality, 2600, Denmark
    Recruiting
  • Regional Hospital Horsens, Department of Medicine
    Horsens, 8700, Denmark
    Recruiting
  • Hospitalsenhed Midt, Medicinsk Diagnostisk Center
    Silkeborg, 8600, Denmark
    Not yet recruiting
  • Svendborg Hospital, Department of Medicine
    Svendborg, 5700, Denmark
    Not yet recruiting
  • Vejle Hospital, Department of Medicine
    Vejle, 7100, Denmark
    Recruiting
07

References and documents

Individual participant data

Plan to share: Yes — All IPD that underlie results in the publications.

Supporting information: Study protocol, Sap, Icf

No publications or documents are linked to this record.

08

Registry details

Key details

Study ID
NCT07552636
Lead sponsor
University of Aarhus
Collaborators
Aarhus University Hospital, Aalborg University Hospital, Svendborg Hospital, Glostrup University Hospital, Copenhagen, Vejle Hospital, Horsens Hospital, Regionshospitalet Silkeborg
Responsible party
Sponsor
First posted
Apr 27, 2026
Start date
May 6, 2026
Primary completion
Mar 2028 (estimated)
Completion
Feb 2029 (estimated)
Last update
Aug 31, 2026

Study contacts

Morten Hansen, Medical Doctor
Contact
mothas@rm.dk
+45 20187463

Oversight

Data monitoring committee
No
FDA-regulated drug
No
FDA-regulated device
No
View the source record on ClinicalTrials.gov ↗

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