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CompletedNCT04387760Updated Oct 26, 2021

Favipiravir vs Hydroxychloroquine vs Control in COVID -19

A Phase 2 interventional study of Hydroxychloroquine and Favipiravir in SARS-CoV 2 and COVID-19, sponsored by Royal College of Surgeons in Ireland - Medical University of Bahrain. Completed at 1 site in Bahrain. Open to participants aged 21 Years and older. Per ClinicalTrials.gov, last updated 2021-10-26.

Sponsored by Royal College of Surgeons in Ireland - Medical University of Bahrain · Phase 2, Interventional, and Treatment

Phase
Phase 2
Study type
Interventional
Enrollment
150
Allocation
Randomized
Ages
21 Years and older
Sex
All
01

Study summary

Hydroxychloroquine is widely used to treat autoimmune diseases. Clinical investigation has found that a high concentration of cytokines were detected in the plasma of critically ill patients infected with SARS-CoV-2, therefore, hydroxychloroquine as anti-inflammatory agents may reduce this response in accord with their use in autoimmune disease where the cytokine response can be reduced.

Favipiravir is an antiviral drug developed in Japan that the data sheet notes that it is a pyrazinecarboxamide derivative with activity against influenza viruses, west nile virus, yellow fever virus, foot and mouth disease virus as well as against flaviviruses, arenaviruses, bunyaviruses and alphaviruses. In February the drug was used for COVID-19 disease in China and was declared effective in treatment, and a report published (in press) comparing Favipiravir with Lopinavir /ritonavir suggested that Favipiravir was superior for prevention of disease progression and viral clearance.

The objective of this pilot study is to compare three arms: hydroxychloroquine; favipiravir; standard care (no specific SARS-CoV-2 treatment) only, in symptomatic patients infected by SARS-CoV-2 in an open label randomized clinical trial. The difference between groups will allow an effect size to be determined for a definitive clinical trial.

Read the detailed description

Coronavirus disease 2019 (COVID-19) is caused by the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2/2019-nCoV) and has developed into a pandemic with serious global public health and economic sequelae. As of June 30, 2020 over 10,000,000 cases have been confirmed worldwide leading to over 500,000 deaths (https://coronavirus.jhu.edu/map.html). Currently no vaccine exists, however chloroquine and hydroxychloroquine have been documented as potentially having antiviral properties with efficacy against COVID-19 disease. Chloroquine is used in the treatment of malaria and amebiasis and is still used in the prophylaxis of malaria. Hydroxychloroquine sulfate is a derivative of Chloroquine that has been demonstrated to be much less (\~40%) toxic than Chloroquine in animals. Hydroxychloroquine is widely used to treat autoimmune diseases, due to its immunomodulatory properties, such as systemic lupus erythematosus and rheumatoid arthritis, with an excellent safety profile. In vitro studies have suggested that their mode of action in COVID-19 disease is blockade of SARS-CoV-2 transport from endosomes to endolysosomes, which appears to be a requirement to release the viral genome. Clinical investigation has found that high concentrations of cytokines are detectable in the plasma of critically ill patients infected with SARS-CoV-2, suggesting that cytokine storm is associated with disease severity; therefore, Chloroquine/ hydroxychloroquine may reduce this response by acting as anti-inflammatory agents in accord with their use in autoimmune disease, where their reduction in cytokine response has been extensively researched and demonstrated.

Favipiravir is an antiviral drug developed in Japan (as noted in the data sheets) that it is a pyrazinecarboxamide derivative with activity against influenza viruses, west nile virus, yellow fever virus, foot and mouth disease virus as well as against flaviviruses (i.e. arenaviruses, bunyaviruses and alphaviruses). Its mode of action is through inhibition of viral RNA-dependent RNA polymerase. In February the drug was used for COVID-19 disease in China and was declared effective in treatment, and a report published (in press) comparing Favipiravir with Lopinavir /ritonavir suggested that Favipiravir was superior for prevention of disease progression and viral clearance.

"The Solidarity Trial" is a global pragmatic clinical trial being undertaken by WHO that aims to explore the efficacy of different treatment modalities for SARS-CoV-2. An application for Bahrain to join the study for collaboration has been made. In "The Solidarity Study" there will be four treatment modalities investigated, including chloroquine phosphate alone, remdesivir, lopinarvir with ritonavir or lopinarvir with ritonavir plus interferon. Favipiravir is not included, and therefore this study will not be replicating features of "The Solidarity Trial" but instead will provide additional and novel findings on favipiravir efficacy.

02

Conditions studied

  • SARS-CoV 2
  • COVID-19

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Keywords

  • Favipiravir
  • hydroxychloroquine
  • COVID-19
03

Who can participate

Ages eligible
21 Years and older
Sexes eligible
All
Accepts healthy volunteers
No

Inclusion criteria

  • Admitted COVID-19 patients being treated as an in-patient at a hospital facility.
  • COVID-19 diagnosis confirmed by PCR nasopharyngeal swab.
  • Study participants must be symptomatic with any COVID-19 symptoms defined by the Bahrain National Protocol
  • Onset of symptoms must be within 10 days prior to enrolment.
  • Study participants must have the ability to give informed consent.
  • Participants must be at minimum 21 years of age.
  • Mild to Moderate COVID-19 disease defined as saturation equals to or more than 93% on room air or PaO2:FiO2 ratio more than 300 on enrolment.

Exclusion criteria

Exclusion Criteria:

  • Severe COVID-19 disease: defined as presence of SpO₂ less than 93% on room air or a PaO₂ to FiO₂ ratio of 300 or lower.
  • Patients on ventilatory support.
  • Cardiac dysfunction that would preclude treatment with hydroxychloroquine:

    1. Patients on medication known to prolong QT segment.
    2. Known history of LQT syndrome.
    3. Acquired QT prolongation at baseline >500ms.
    4. AV block.
    5. Bundle Branch Block.
    6. Known history of Cardiomyopathy, Pulmonary Hypertension, or Sick Sinus Syndrome.
    7. History of ventricular tachyarrhythmia.
    8. Patients with implantable cardioverter-defibrillator (ICD).
    9. Patients with a baseline bradycardia of less than 50 beats per minute.
  • Renal dysfunction (estimated glomerular filtration rate less than 30ml/min).
  • Hepatic dysfunction defined as:

    1. Transaminitis more than three times the upper limit of normal or
    2. Chronic liver disease of Child Pugh Class B or higher.
  • Gout or a history of gout
  • Patients that are pregnant or breastfeeding.
  • Patients with a known allergy to an intervention medication.
  • Patients who receive any of the study medications prior to randomization
  • Patient with G6PD
  • Readmission due to COVID19 disease.
  • Participants in any other COVID-19 disease trial.
  • Patients on immunosuppressants, HIV patients, cancer patients who received chemotherapy within the past 6 months, or who are on chronic oral steroids.
  • Patients unable to give informed consent.
04

Study design

Phase
Phase 2
Primary purpose
Treatment
Allocation
Randomized
Intervention model
Parallel assignment
Masking
None (open label)
Enrollment
150 participants (actual)

Study arms

  • Experimental
    Hydroxychloroquine

    Hydroxychloroquine is widely used to treat autoimmune diseases, due to its immunomodulatory properties, such as systemic lupus erythematosus and rheumatoid arthritis, with an excellent safety profile. In vitro studies have suggested that their mode of action in COVID-19 disease is blockade of SARS-CoV-2 transport from endosomes to endolysosomes, which appears to be a requirement to release the viral genome.

    Drug: Hydroxychloroquine · Other: Routine care for COVID-19 patients

  • Experimental
    Favipiravir

    Favipiravir is an antiviral drug that it is a pyrazinecarboxamide derivative with activity against influenza viruses, west nile virus, yellow fever virus, foot and mouth disease virus as well as against flaviviruses (i.e. arenaviruses, bunyaviruses and alphaviruses).

    Drug: Favipiravir · Other: Routine care for COVID-19 patients

  • Active comparator
    Standard clinical care

    Supportive care according to local guidelines

    Other: Routine care for COVID-19 patients

Interventions

  • DrugHydroxychloroquine

    400mg BID PO day 1 then 200mg BID PO from day 2 to day 10. In addition to Hydroxychloroquine all patients will receive the standard care (according to local Bahrain COVID19 guidelines). Any patient who is fit for discharge, can be discharged and medications will be stopped on discharge.

    Also known as: Hydroxychloroquine sulfate, Plaquenil

  • DrugFavipiravir

    1600mg BID PO day 1600mg BID PO day 2 to day 10. In addition to Favipiravir all patients will receive the standard care (according to local Bahrain COVID19 guidelines). Any patient who is fit for discharge, can be discharged and medications will be stopped on discharge.

    Also known as: Avigan, T-705, favipira, favilavir

  • OtherRoutine care for COVID-19 patients

    Supportive care according to local guidelines

    Also known as: Standard clinical care

05

What researchers measure

Primary outcomes

  1. Primary outcome is the Medial clinical scale at end of study follow up

    Median clinical scale at end of study follow up (day 14 or on discharge/death, whichever is earlier)

    Time frame: Until discharge, death or for a maximum of 30 days or readmission

Secondary outcomes

  1. Requirement of Escalation of Respiratory Support

    Implementation of escalation of Respiratory Support

    Time frame: Until discharge, death or for a maximum of 14 days or readmission

  2. Adverse effects(cardiac, renal, hepatic, hypoglycaemia (defined as RBS <3.9 mmol/L))

    Monitor and document all adverse effects during therapy

    Time frame: Until discharge,death or for a maximum of 14 days or readmission

  3. Requirement of ICU Admission

    Deterioration of clinical condition requiring ICU admission

    Time frame: Until discharge, death or for a maximum of 14 days or readmission

  4. Mortality rate

    30 days Mortality rate due to COVID-19

    Time frame: Mortality will be collected up to 30 days

  5. Readmission rate

    30 days readmission rate will be captured

    Time frame: Readmission will be collected up to 30 days from start of the study

  6. Daily National Early Warning (NEWS) 2 Score

    Daily NEWS 2 will be calculated which is a tool that improves the detection and response to clinical deterioration in adult patients and is a key element of patient safety and improving patient outcomes

    Time frame: Until discharge, death or for a maximum of 14 days

  7. Daily Sequential Organ Failure Assessment (SOFA) score

    Daily SOFA score will be calculated which can identify the critical point at which patients exhibit the highest degree of organ dysfunction

    Time frame: Until discharge, death or for a maximum of 14 days

  8. Change in Laboratory indices

    Determination of the change in D-dimer, ratio of Lymphocyte to Neutrophil, lactate before and after treatments as a measure of disease activity

    Time frame: Until discharge, death or for a maximum of 14 days

  9. Discharge and Length of Hospital Stay

    Patients will be followed during their hospital stay until discharge

    Time frame: Until discharge, death or for a maximum of 14 days

  10. QT prolongation

    Determination of the change in QT prolongation, before and after treatments as a measure of disease activity

    Time frame: Until discharge, death or for a maximum of 14 days or readmission

  11. Cardiac arrythmia (fatal and non fatal)

    Detection of Cardiac arrythmia (fatal and non fatal), before and after treatments as a measure of disease activity

    Time frame: Until discharge, death or for a maximum of 14 days or readmission

  12. Viral clearance

    Viral clearance defined as a single negative SARS-CoV2 PCR nasopharyngeal swab

    Time frame: until discharge, death or for a maximum of 30 days

06

Study locations

1 site
  • Royal College of Surgeons in Ireland - Bahrain
    Manama, Bahrain
07

References and documents

Publications

  • McChesney EW. Animal toxicity and pharmacokinetics of hydroxychloroquine sulfate. Am J Med. 1983 Jul 18;75(1A):11-8. doi: 10.1016/0002-9343(83)91265-2. PubMed 6408923 ↗
  • Liu J, Cao R, Xu M, Wang X, Zhang H, Hu H, Li Y, Hu Z, Zhong W, Wang M. Hydroxychloroquine, a less toxic derivative of chloroquine, is effective in inhibiting SARS-CoV-2 infection in vitro. Cell Discov. 2020 Mar 18;6:16. doi: 10.1038/s41421-020-0156-0. eCollection 2020. No abstract available. PubMed 32194981 ↗
  • Huang C, Wang Y, Li X, Ren L, Zhao J, Hu Y, Zhang L, Fan G, Xu J, Gu X, Cheng Z, Yu T, Xia J, Wei Y, Wu W, Xie X, Yin W, Li H, Liu M, Xiao Y, Gao H, Guo L, Xie J, Wang G, Jiang R, Gao Z, Jin Q, Wang J, Cao B. Clinical features of patients infected with 2019 novel coronavirus in Wuhan, China. Lancet. 2020 Feb 15;395(10223):497-506. doi: 10.1016/S0140-6736(20)30183-5. Epub 2020 Jan 24. Erratum In: Lancet. 2020 Feb 15;395(10223):496. doi: 10.1016/S0140-6736(20)30252-X. PubMed 31986264 ↗
  • Delang L, Abdelnabi R, Neyts J. Favipiravir as a potential countermeasure against neglected and emerging RNA viruses. Antiviral Res. 2018 May;153:85-94. doi: 10.1016/j.antiviral.2018.03.003. Epub 2018 Mar 7. PubMed 29524445 ↗
  • Dong L, Hu S, Gao J. Discovering drugs to treat coronavirus disease 2019 (COVID-19). Drug Discov Ther. 2020;14(1):58-60. doi: 10.5582/ddt.2020.01012. PubMed 32147628 ↗
  • Cai Q, Yang M, Liu D, Chen J, Shu D, Xia J, Liao X, Gu Y, Cai Q, Yang Y, Shen C, Li X, Peng L, Huang D, Zhang J, Zhang S, Wang F, Liu J, Chen L, Chen S, Wang Z, Zhang Z, Cao R, Zhong W, Liu Y, Liu L. Experimental Treatment with Favipiravir for COVID-19: An Open-Label Control Study. Engineering (Beijing). 2020 Oct;6(10):1192-1198. doi: 10.1016/j.eng.2020.03.007. Epub 2020 Mar 18. PubMed 32346491 ↗
  • Gautret P, Lagier JC, Parola P, Hoang VT, Meddeb L, Mailhe M, Doudier B, Courjon J, Giordanengo V, Vieira VE, Tissot Dupont H, Honore S, Colson P, Chabriere E, La Scola B, Rolain JM, Brouqui P, Raoult D. RETRACTED: Hydroxychloroquine and azithromycin as a treatment of COVID-19: results of an open-label non-randomized clinical trial. Int J Antimicrob Agents. 2020 Jul;56(1):105949. doi: 10.1016/j.ijantimicag.2020.105949. Epub 2020 Mar 20. PubMed 32205204 ↗
  • Birkett MA, Day SJ. Internal pilot studies for estimating sample size. Stat Med. 1994 Dec 15-30;13(23-24):2455-63. doi: 10.1002/sim.4780132309. PubMed 7701146 ↗
  • Senn SJ. Covariate imbalance and random allocation in clinical trials. Stat Med. 1989 Apr;8(4):467-75. doi: 10.1002/sim.4780080410. PubMed 2727470 ↗
  • Senn S. Testing for baseline balance in clinical trials. Stat Med. 1994 Sep 15;13(17):1715-26. doi: 10.1002/sim.4780131703. PubMed 7997705 ↗
  • Knol MJ, Groenwold RH, Grobbee DE. P-values in baseline tables of randomised controlled trials are inappropriate but still common in high impact journals. Eur J Prev Cardiol. 2012 Apr;19(2):231-2. doi: 10.1177/1741826711421688. No abstract available. PubMed 22512015 ↗
  • AlQahtani M, Kumar N, Aljawder D, Abdulrahman A, Mohamed MW, Alnashaba F, Fayyad MA, Alshaikh F, Alsahaf F, Saeed S, Almahroos A, Abdulrahim Z, Otoom S, Atkin SL. Randomized controlled trial of favipiravir, hydroxychloroquine, and standard care in patients with mild/moderate COVID-19 disease. Sci Rep. 2022 Mar 23;12(1):4925. doi: 10.1038/s41598-022-08794-w. Erratum In: Sci Rep. 2022 Sep 26;12(1):16052. doi: 10.1038/s41598-022-20899-w. PubMed 35322077 ↗

Individual participant data

Plan to share: Yes — Monitoring, audits, and REC review will be permitted and provide direct access to source data and documents. The Lead PI and the researchers assigned by him will have access to the stored data/specimens. Only the Lead PI and the researchers assigned working on this study will be eligible to obtain the data/specimens from the participants during data collection.

Supporting information: Study protocol, Sap, Csr, Analytic code

08

Registry details

Key details

Study ID
NCT04387760
Lead sponsor
Royal College of Surgeons in Ireland - Medical University of Bahrain
Collaborators
Ebrahim Khalil Kanoo Community Medical Center, Hereditary blood Disorder Centre - Salmaniya Medical Complex, Mohammed Bin Khalifa Bin Sulman Al Khalifa Cardiac Centre, Awali, Jidhafs COVID-19 Centre, Sitra FICU, Salmaniya Medical Complex- 6th Floor, Salmaniya Medical Complex- Helipad
Responsible party
Sponsor
First posted
May 14, 2020
Start date
Aug 11, 2020
Primary completion
Mar 6, 2021
Completion
Apr 7, 2021
Last update
Oct 26, 2021

Study contacts

Manaf Al Qahtani, Dr.
principal investigator · Royal College of Surgeons in Ireland - Bahrain

Oversight

Data monitoring committee
Yes
FDA-regulated drug
No
FDA-regulated device
No
View the source record on ClinicalTrials.gov ↗

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