CClinicalTrials.gg
Status unknownNCT02810717Updated Aug 16, 2018

Effects of TBS on 5-HT1A Receptor Binding

An interventional study of theta-burst stimulation using a MagPro X1000 and sham stimulation using a MagPro X1000 in Treatment Resistant Depression, sponsored by Rupert Lanzenberger. Status unknown at 1 site in Austria. Open to participants aged 18 Years to 65 Years. Per ClinicalTrials.gov, last updated 2018-08-16.

Sponsored by Rupert Lanzenberger · Not applicable, Interventional, and Treatment

The sponsor has not verified this record recently (last verified Aug 2018), so the status shown — last known as Recruiting — may be out of date.
Phase
Not applicable
Study type
Interventional
Enrollment
80
Allocation
Randomized
Ages
18 Years to 65 Years
Sex
All
01

Study summary

Background:

Theta-burst stimulation (TBS), a form of repetitive transcranial magnetic stimulation (rTMS) holds promise as an effective treatment for treatment resistant depression (TRD). rTMS has been linked to neuroplastic changes as shown using magnetic resonance imaging (MRI) and positron emission tomography (PET). Alterations in serotonin-1A receptor expression (5-HT1A) have been linked to major depression. Moreover, changes in 5-HT1A receptor binding - observed after pharmacological treatment, as well as after electroconvulsive therapy - has been linked to neuronal adaptations in response to these antidepressant treatments.

Objectives of the study:

Here, the aim is to investigate the effects of TBS over left and right dorsolateral prefrontal cortex on the 5-HT1A receptor binding in patients with TRD using PET. In addition, effects of iTBS on brain structure and function will be determined using functional, structural and perfusion MRI.

Study population:

80 patients with TRD who maintain their original medication regimen will be recruited.

Study design:

Longitudinal, randomized and double-blind clinical trial. 40 patients will receive active TBS, 40 patients will receive sham TBS for treatment duration of three weeks. Before and after three weeks of treatment, patients will be scanned using MRI and PET with the highly specific and selective radiotracer [carbonyl-11C]WAY100635. A follow-up visit and final examination will be performed 2 and 4 weeks after treatment for the active TBS group, respectively. Patients in the sham TBS arm will receive active TBS treatment immediately after the second MRI and PET scan.

Relevance and implications of the study:

This will be the worldwide first multimodal imaging study to investigate the effects of TBS on serotonin-1A receptor binding in TRD using PET. Thus, the study will add crucial knowledge to the existing literature on the effects of TMS on brain structure and function, related to antidepressant efficacy. Moreover, by combining molecular imaging of serotonergic neurotransmission with structural and functional MRI, the proposed study will increase the investigators knowledge on the serotonergic role in shaping brain morphology, microstructure and structural/functional connectivity. Taken together, the study has the potential to contribute to the development of personalized treatment, the reduction of personal suffering and the reduction of costs and occupational disability.

02

Conditions studied

  • Treatment Resistant Depression
03

Who can participate

Ages eligible
18 Years to 65 Years
Sexes eligible
All
Accepts healthy volunteers
No

Inclusion criteria

  • DSM-5 diagnosis of single or recurrent major depression
  • HAMD-17 total score of ≥ 18 and a Clinical Global Impression Scale (CGI-S) of ≥ 4
  • Failure of at least two adequate antidepressant treatments
  • Age 18-65 years
  • Right-handedness (assessed with the Edinburgh Handedness Inventory)

Exclusion criteria

Exclusion Criteria:

  • Seizures in medical history
  • Lifetime medical history of major systemic illness, neurological disorders and previous brain injuries
  • Ferromagnetic implants, cardiac pacemaker, deep brain stimulation and other common MRI exclusion criteria
  • Lifetime history of psychotic disorders or current psychotic symptoms
  • Substance abuse or dependence within the last 3 months
  • Borderline personality disorder (based on DSM-5 criteria)
  • Pregnancy
  • Active suicidal intent
  • Benzodiazepines other than Lorazepam > 2mg/d or any dose of an anticonvulsant
  • for participants who participated in an earlier neuroimaging study using ionizing radiation, the total radiation exposure dose of 20 mSv over the last 10 years must not be exceeded, as specified in the legislation on radiation protection.
  • failure to comply with the study protocol or to follow the instructions of the investigating team
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Study design

Phase
Not applicable
Primary purpose
Treatment
Allocation
Randomized
Intervention model
Parallel assignment
Masking
Double (Participant, Care provider)
Enrollment
80 participants (estimated)

Study arms

  • Experimental
    active TBS

    40 patients with TRD will receive active theta-burst stimulation using a MagPro X1000 between the two PET measurements

    Device: theta-burst stimulation using a MagPro X1000

  • Sham comparator
    sham TBS

    40 patients with TRD will receive sham stimulation using a MagPro X1000 between the two PET measurements. After the second PET scan they will receive active TBS

    Device: sham stimulation using a MagPro X1000

Interventions

  • Devicetheta-burst stimulation using a MagPro X1000

    TBS over left and right dorsolateral prefrontal cortex for a period of three weeks. iTBS over left DLPFC: 3-pulse 50 Hz bursts will be given every 200ms (at 5 Hz) in 2-second trains with an inter-train interval of 8 seconds. Trains will be repeated 20 to reach a total number of 600 pulses per session. cTBS over right DLPFC: cTBS will comprise uninterrupted bursts to reach a total number of 600 pulses per session. Two sessions per day, separated by 60 minutes; 30 Sessions in total over 3 weeks.

    Also known as: repetitive transcranial magnetic stimulation

  • Devicesham stimulation using a MagPro X1000

    Sham TBS with the coil set at 45° against the skull will be performed over left and right dorsolateral prefrontal cortex for a period of three weeks

    Also known as: sham transcranial magnetic stimulation

05

What researchers measure

Primary outcomes

  1. Regional 5-HT1A receptor binding

    5-HT1A receptor binding using the radioligand \[carbonyl-11C\]WAY100635

    Time frame: before and after 3 weeks of TBS treatment

Secondary outcomes

  1. Regional white matter microstructure using DWI-TBSS

    The analysis will be performed using tract-based spatial statistics

    Time frame: before and after 3 weeks of TBS treatment

  2. Regional white matter microstructure using DWI-Tractography

    Tractography will be performed

    Time frame: before and after 3 weeks of TBS treatment

  3. Regional grey matter volume using MRI

    The analysis will be done using voxel-based morphometry

    Time frame: before and after 3 weeks of TBS treatment

  4. Regional brain perfusion

    Regional brain perfusion will be evaluated using Arterial Spin Labeling, ASL

    Time frame: before and after 3 weeks of TBS treatment

  5. Functional connectivity at rest and during tasks

    Functional connectivity will be evaluated using resting state and task fMRI

    Time frame: before and after 3 weeks of TBS treatment

Other outcomes

  1. Depression score using the Hamilton Depression Rating Scale

    Depression will be evaluated using the Hamilton Depression Rating Scale

    Time frame: before and after 3 weeks of TBS treatment

  2. Depression score using the Beck Depression Inventory

    Depression will be evaluated using the Beck Depression Inventory

    Time frame: before and after 3 weeks of TBS treatment

  3. Global physical activity

    assessed using the WHO global physical assessment GPAQ

    Time frame: before and after 3 weeks of TBS treatment

06

Study locations

1 of 1 sites recruiting
  • Department of Psychiatry and Psychotherapy, Medical University of Vienna
    Vienna, A-1090, Austria
    Recruiting
07

References and documents

Individual participant data

Plan to share: Yes — A database that also includes data from this study will be created

08

Registry details

Key details

Study ID
NCT02810717
Lead sponsor
Rupert Lanzenberger
Responsible party
Rupert Lanzenberger (Assoc.-Prof. PD MD, Medical University of Vienna) — Sponsor-investigator
First posted
Jun 23, 2016
Start date
Dec 2016
Primary completion
Nov 2019 (estimated)
Completion
Nov 2019 (estimated)
Last update
Aug 16, 2018

Study contacts

Rupert Lanzenberger, MD
Contact
rupert.lanzenberger@meduniwien.ac.at
+43 40400 35760
Georg Kranz, PhD
Contact
georg.kranz@meduniwien.ac.at
+43 40400 38250
Siegfried Kasper, MD
principal investigator · Department of Psychiatry and Psychotherapy, Medical University of Vienna

Oversight

Data monitoring committee
No
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